Results 211 to 220 of about 18,386 (273)

When the Position of Pendant Groups Makes the Difference in G-Quadruplex Behavior: The Case of Bis-Conjugated Thrombin-Binding Aptamers. [PDF]

open access: yesJ Chem Inf Model
Platella C   +6 more
europepmc   +1 more source

Measurement of Active Thrombin Bound to Circulating D‐Dimers as a Sensitive Biomarker for Prothrombotic Conditions

open access: yesJournal of Clinical Laboratory Analysis, Volume 40, Issue 11, June 2026.
Scheme of preparation of D‐dimers and evaluation of D‐dimer‐bound thrombin activity. Thrombin bound to fibrin participates in ongoing fibrinogen transformation and its inhibition is impeded. Our detection system may aid in identifying patients at risk of recurrent thrombotic events or complications, regardless of the origin of the acute health problem.
Jana Stikarova   +15 more
wiley   +1 more source

A review of the protection mechanisms of ginsenoside CK on cardiovascular disease and stroke. [PDF]

open access: yesFront Pharmacol
Chan HT   +9 more
europepmc   +1 more source

Platelet‐Rich Fibrin Induces New Bone Formation to Promote Extreme Lateral Interbody Fusion of Spine in Rabbits

open access: yesJOR SPINE, Volume 9, Issue 2, June 2026.
Preparation process of PRF and its promotion of intervertebral fusion. The rabbit lateral fusion model proves that PRF can promote the intervertebral fusion rate of allograft bone. In vitro experiments can be observed that PRF promotes the osteogenic expression of BMSCS.
Weijian Wang   +5 more
wiley   +1 more source

Anion‐Specific Mechanisms in Fibrinogen Self‐Assembly: Contrasting Effects of Phosphates and Chlorides in Nanofiber Formation

open access: yesMacromolecular Bioscience, Volume 26, Issue 6, June 2026.
Fibrinogen self‐assembly follows distinct anion‐directed pathways. Phosphate promotes ordered nucleation and elongation into interconnected nanofibrous networks, whereas chloride induces disordered porous aggregates. Specific phosphate retention within the fibers suggests direct ion–protein interactions during fibrillogenesis.
Antoine Eyram Kwame   +5 more
wiley   +1 more source

Pathogenic Mechanisms of V. vulnificus and Its Role in the Development of Sepsis

open access: yesMicrobiologyOpen, Volume 15, Issue 3, June 2026.
Sorafenib attenuates urethral scar formation by inhibiting β‐catenin signaling in fibroblasts, which downregulates the SLC7A11/GPX4 antioxidant axis and triggers ferroptosis. This β‐catenin–ferroptosis coupling effectively suppresses fibrogenesis, highlighting sorafenib as a promising drug‐repurposing candidate for urethral stricture.
Zhongying Yu   +7 more
wiley   +1 more source

Regulation of platelet contractility by agonists present across a thrombus. [PDF]

open access: yesBlood Adv
Hiebner DW   +9 more
europepmc   +1 more source

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