Results 151 to 160 of about 2,296 (190)

Proteomic Profiling of Pre- and Post-Surgery Saliva of Glioblastoma Patients: A Pilot Investigation. [PDF]

Int J Mol Sci
Muntiu A, Moresi F, Vincenzoni F, Rossetti DV, Iavarone F, Messana I, Castagnola M, La Rocca G, Mazzucchi E, Olivi A, Urbani A, Sabatino G, Desiderio C.   +12 more
europepmc   +1 more source

The role of miRNA regulation in cancer progression and drug resistance [PDF]

, 2012
Joshi, Tejal
core  

Bridging the gap for diverse applications of parasites as advanced cancer therapeutics: current progress and future directions. [PDF]

Infect Agent Cancer
Eissa MM, El-Faham MH, El Skhawy N.
europepmc   +1 more source

ATG9 vesicles are a subtype of intracellular nanovesicle

Fesenko M, Moore DJ, Ewbank P, Courthold E, Royle SJ.   +4 more
europepmc   +1 more source

Tumor protein D52 (TPD52) and cancer—oncogene understudy or understudied oncogene?

Tumor Biology, 2014
The Tumor protein D52 (TPD52) gene was identified nearly 20 years ago through its overexpression in human cancer, and a substantial body of data now strongly supports TPD52 representing a gene amplification target at chromosome 8q21.13. This review updates progress toward understanding the significance of TPD52 overexpression and targeting, both in ...
Jennifer Anne Byrne, Yuyan Chen, Robert K Bright   +2 more
exaly   +3 more sources

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Circ_0060551 promotes the migration and invasion of cervical cancer by upregulating TPD52

American Journal of Reproductive Immunology, 2022
AbstractProblemCervical cancer has been recognized as the second most common cancer in women worldwide. Previous studies have reported that some circular RNAs (circRNAs) can influence the progression of cervical cancer. However, more researches are still required to unveil the underlying mechanism of how circRNAs regulate the progression of such cancer.
Qi, Shi, Yuanyuan, Ma, Xiaojie, Zhang, Chunxia, Yin   +3 more
openaire   +2 more sources

Acetylation-dependent regulation of TPD52 isoform 1 modulates chaperone-mediated autophagy in prostate cancer

Autophagy, 2021
Aberrant chaperone-mediated autophagy (CMA) activation has been suggested as a tumorigenesis-promoting event in various cancers, although its roles in prostate cancer (PCa) remain elusive. Emerging evidence indicates that TPD52 isoform 1, a prostate-specific and androgen-responsive gene, contributes to the malignant progression of PCa.
Yizeng Fan, Tao Hou, Jin Zeng
exaly   +3 more sources

MiR-125b-5p/TPD52 Axis Affects Proliferation, Migration and Invasion of Breast Cancer Cells

Molecular Biotechnology, 2022
Aberrant gene expression caused by miRNAs disorders plays a relevant role in multiple steps of tumorigenesis. In this attempt, we studied the functional role of miR-125b-5p and TPD52 in breast cancer. TPD52 mRNA and miR-125b-5p levels were assessed via qRT-PCR, and TPD52 protein level was analyzed via western blot.
Yuezhen Wang, Jun Fang
exaly   +3 more sources

Overexpression, Amplification, and Androgen Regulation of TPD52 in Prostate Cancer

Cancer Research, 2004
Abstract Gains in the long arm of chromosome 8 (8q) are believed to be associated with poor outcome and the development of hormone-refractory prostate cancer. Based on a meta-analysis of gene expression microarray data from multiple prostate cancer studies (D. R.
Mark A, Rubin, Sooryanarayana, Varambally, Rameen, Beroukhim, Scott A, Tomlins, Daniel R, Rhodes, Pamela L, Paris, Matthias D, Hofer, Martina, Storz-Schweizer, Rainer, Kuefer, Jonathan A, Fletcher, Bae-Li, Hsi, Jennifier A, Byrne, Kenneth J, Pienta, Colin, Collins, William R, Sellers, Arul M, Chinnaiyan   +15 more
openaire   +2 more sources

The Role of the Coiled-Coil Motif in Interactions Mediated by TPD52

Biochemical and Biophysical Research Communications, 2001
TPD52 (D52)-like proteins are small coiled-coil motif-bearing proteins first identified through their expression in human breast carcinoma that mutually interact in hetero- and homomeric fashions. However, it has been unclear whether the coiled-coil motif is sufficient, or even necessary, for these interactions to occur.
P, Sathasivam, A M, Bailey, M, Crossley, J A, Byrne   +3 more
openaire   +2 more sources