Transforming growth factor β1 inhibition protects from noise-induced hearing loss [PDF]
Frontiers in Aging Neuroscience, 2015 Excessive exposure to noise damages the principal cochlear structures leading to hearing impairment. Inflammatory and immune responses are central mechanisms in cochlear defensive response to noise but, if unregulated, they contribute to inner ear damage
Silvia eMurillo-Cuesta +19 more
doaj +5 more sources
Role of transforming growth factor-β1 in breast carcinogenesis [PDF]
Archive of Oncology, 2006 The main objective of this presentation is to review current knowledge regarding molecular mechanisms of Transforming Growth Factor-β1(TGF-ß1) action in breast carcinogenesis.
Ivanović Vesna +3 more
doaj +3 more sources
Comparison of Transforming Growth Factor-Beta 1 Concentration in Preeclampsia and Normal Pregnancy Women [PDF]
Indonesian Biomedical Journal, 2017 BACKGROUND: According to the theory of endothelial dysfunction, the pathogenesis of preeclampsia is associated with the imbalance of angiogenic and anti-angiogenic factors.
Yusrawati Yusrawati +2 more
doaj +2 more sources
Corrigendum to: Transforming growth factor β1 inhibition protects from noise-induced hearing loss
Frontiers in Aging Neuroscience, 2015 Unfortunately, the second author affiliation in the originally published article was incorrectly stated as being “Centre for Biomedical Network Research (CIBER), Institute of Health Carlos III (ISCIII), Madrid, Spain.” The correct author's affiliation is “Centre for Biomedical Network Research on Rare Diseases (CIBERER), Institute of Health Carlos ...
Silvia eMurillo-Cuesta +19 more
doaj +4 more sources
Enhanced Expression of Integrin αvβ3 Induced by TGF-β Is Required for the Enhancing Effect of Fibroblast Growth Factor 1 (FGF1) in TGF-β-Induced Epithelial-Mesenchymal Transition (EMT) in Mammary Epithelial Cells. [PDF]
, 2015 Epithelial-to-mesenchymal transition (EMT) plays a critical role in cancer metastasis, and is regulated by growth factors such as transforming growth factor β (TGF-β) and fibroblast growth factors (FGF) secreted from the stromal and tumor cells. However,
Hamada, Yoshinosuke +7 more
core +10 more sources
Transforming Growth Factor-β1 T869C Gene Polymorphism Is Associated with Acquired Sick Sinus Syndrome via Linking a Higher Serum Protein Level. [PDF]
PLoS ONE, 2016 Familial sick sinus syndrome is associated with gene mutations and dysfunction of ion channels. In contrast, degenerative fibrosis of the sinus node tissue plays an important role in the pathogenesis of acquired sick sinus syndrome.
Jan-Yow Chen +5 more
doaj +1 more source
Romanian Journal of Laboratory Medicine, 2022 Background: To study the expressions of transforming growth factor-β1 (TGF-β1) and vascular endothelial growth factor (VEGF) in patients with acute myeloid leukemia (AML) and their values for prognosis.
Xu Yan, Yu Xianqiu, Zhang Xinlong
doaj +1 more source
Platelet-derived transforming growth factor-β1 promotes keratinocyte proliferation in cutaneous wound healing. [PDF]
, 2020 Platelets are a recognised potent source of transforming growth factor-β1 (TGFβ1), a cytokine known to promote wound healing and regeneration by stimulating dermal fibroblast proliferation and extracellular matrix deposition.
Chong, DLW +7 more
core +3 more sources
Correlation of Expression Transforming Growth Factor-β1, E-cadherin, and Ki-67 in Meningiomas
Open Access Macedonian Journal of Medical Sciences, 2020 BACKGROUND: Meningioma is the most common primary intracranial tumors in adults, accounts for 36% of total intracranial tumors. Obtaining the clinicopathological characteristics of patients with meningioma and investigating, the association between signaling pathways with disease progression could provide a basis for therapeutic development.AIM: This ...
Apriyanto, Apriyanto +3 more
openaire +2 more sources
Frontiers in Pharmacology, 2020 Idiopathic pulmonary fibrosis is a chronic, progressive, and lethal lung disease of unknown etiology. Antifibrotic drugs, including pirfenidone, are currently used for the treatment of the disease.
Tomohito Okano +21 more
doaj +1 more source