Results 171 to 180 of about 25,325 (230)

Hydration-Controlled Proton Transport in Respiratory Complex I. [PDF]

J Am Chem Soc
Choi JH, Voth GA.
europepmc   +1 more source

Characterization of the complete mitochondrial genome and phylogenetic analysis of <i>Tetraselmis marina</i> (Cienkowski) R.E.Norris, Hori & Chihara 1980. [PDF]

Mitochondrial DNA B Resour
Cheng C, Chen CH, Yang F.
europepmc   +1 more source

Temporal Wheat Proteome Remodeling by Deoxynivalenol Reveals Novel Detoxification Signatures and Strategies Across Cultivars. [PDF]

Mol Cell Proteomics
Buchanan R, Shaban K, Liu B, Chan N, Serajazari M, Geddes-McAlister J.   +5 more
europepmc   +1 more source

Ischaemia-Reperfusion Injury in Organ Transplantation: Role of Coenzyme Q10. [PDF]

J Clin Med
Mantle D, Cufflin N, Purcell TT, Hargreaves IP.   +3 more
europepmc   +1 more source

A systems-level view of metabolic impact of copper and methane valorization potential in <i>Methylosinus trichosporium</i> OB3b. [PDF]

Metab Eng Commun
Yang J, Semrau JD.
europepmc   +1 more source

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Solubilized ubiquinol for preserving corneal function

Biomaterials, 2021
Defective cellular metabolism, impaired mitochondrial function, and increased cell death are major problems that adversely affect donor tissues during hypothermic preservation prior to transplantation. These problems are thought to arise from accumulated reactive oxygen species (ROS) inside cells.
Youssef W Naguib, Sanjib Saha, Timothy M Acri   +2 more
exaly   +3 more sources

Mitochondrial ubiquinol oxidation is necessary for tumour growth [PDF]

Nature, 2020
The mitochondrial electron transport chain (ETC) is necessary for tumour growth1-6 and its inhibition has demonstrated anti-tumour efficacy in combination with targeted therapies7-9. Furthermore, human brain and lung tumours display robust glucose oxidation by mitochondria10,11. However, it is unclear why a functional ETC is necessary for tumour growth
Inmaculada C Martinez-Reyes, Hyewon Kong, Gregory S Mcelroy   +2 more
exaly   +3 more sources

AIFM2 blocks ferroptosis independent of ubiquinol metabolism

Biochemical and Biophysical Research Communications, 2020
Ferroptosis is a multi-step regulated cell death that is characterized by excessive iron accumulation and lipid peroxidation. Cancer cells can acquire resistance to ferroptosis by the upregulation of anti-ferroptotic proteins or by the downregulation of pro-ferroptotic proteins.
Enyong Dai, Rui Kang, Daolin Tang
exaly   +3 more sources