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IL-33 blockade in asthma: a story of hype, hope and hindsight. [PDF]
ERJ Open ResGeoras SN, Bourdin A.
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A neuroimmune axis linking S100A8/9 to itch sensitization in both bullous pemphigoid and atopic dermatitis. [PDF]
JID InnovLawson K +8 more
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UDP-Glucose-P2Y14 Receptor Signaling Enhances the Responsiveness of Hematopoietic Stem/Progenitor Cells to Chemoattractants by Facilitating Membrane Lipid Raft Formation and Nlrp3 Inflammasome Activation. [PDF]
Stem Cell Rev RepAdamiak M +9 more
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HMGB1-dependent signaling in the regulation of mast cell activity during inflammation. [PDF]
Front ImmunolAgier J +6 more
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Alarmins in autoimmune diseases
Autoimmunity Reviews, 2022Alarmins are endogenous, constitutively expressed, chemotacting and immune activating proteins or peptides released because of non-programmed cell death (i.e. infections, trauma, etc). They are considered endogenous damage-associated molecular patterns (DAMPs), able to induce a sterile inflammation.
Danieli, Maria Giovanna +7 more
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[Alarmins and anti-alarmin biologics in asthma].
Tuberkuloz ve toraks, 2018Alarmins are endogenous, constitutively expressed, chemotactic, and immune activating proteins/peptides that are released as a result of degranulation, cell injury or death, or in response to immune induction. Alarmins are involved in a variety of processes including antimicrobial gene expression regulation, cellular homeostasis, wound healing ...
Begüm, Görgülü, Sevim, Bavbek
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