RhoA/ROCK Pathway Activation is Regulated by AT1 Receptor and Participates in Smooth Muscle Migration and Dedifferentiation via Promoting Actin Cytoskeleton Polymerization. [PDF]
Qi Y +5 more
europepmc +1 more source
Le Système Rénine Angiotensine tient une place prépondérante au sein de la circulation cérébrale. Les Antagonistes des Récepteurs AT1 à l'Angiotensine II (ARAII) ont prouvé leur efficacité dans la prévention de l'Accident Vasculaire Cérébral (AVC ...
Foulquier, Sébastien
core
An Open-Label, Non-randomized, Drug-Repurposing Study to Explore the Clinical Effects of Angiotensin II Type 1 (AT1) Receptor Antagonists on Anxiety and Depression in Parkinson's Disease. [PDF]
Bordet S +6 more
europepmc +1 more source
Age-Related Effects of AT1 Receptor Antagonist Losartan on Cognitive Decline in Spontaneously Hypertensive Rats. [PDF]
Tchekalarova J +2 more
europepmc +1 more source
AZ AT1-R G-FEHÉRJE FÜGGETLEN JELÁTVITELÉNEK VIZSGÁLATA = G PROTEIN-INDEPENDENT SIGNALING OF AT1-R
Az angiotenzin II (AngII) a renin-angiotenzin rendszer fő szabályozója és nemcsak normál élettani szabályozási folyamatokban, hanem számos kórélettani folyamatban is szerepet játszik.
Balla, András
core
Autoantibodies against AT1 Receptor Contribute to Vascular Aging and Endothelial Cell Senescence. [PDF]
Wang M +9 more
europepmc +1 more source
Increase in body weight is lowered when mice received fecal microbiota transfer from donor mice treated with the AT1 receptor antagonist telmisartan. [PDF]
Freschi ML +6 more
europepmc +1 more source
Endogenous AT1 receptor-protein kinase C activity in the hypothalamus augments glutamatergic input and sympathetic outflow in hypertension. [PDF]
Ma H, Chen SR, Chen H, Pan HL.
europepmc +1 more source
Insights Into the Role of Angiotensin-II AT<sub>1</sub> Receptor-Dependent β-Arrestin Signaling in Cardiovascular Disease. [PDF]
Mathieu NM +3 more
europepmc +1 more source
AT1-receptor blockade: Protective effects of irbesartan in cardiomyocytes under hypoxic stress. [PDF]
Boccellino M +7 more
europepmc +1 more source

