Results 31 to 40 of about 1,985,012 (310)

Effects and Mechanisms of Exosomes from Different Sources in Cerebral Ischemia

open access: yesCells, 2022
Cerebral ischemia refers to the symptom of insufficient blood supply to the brain. Cells of many different origins participate in the process of repairing damage after cerebral ischemia occurs, in which exosomes secreted by the cells play important roles.
Ruoxi Xie   +4 more
doaj   +1 more source

Review on herbal medicine on brain ischemia and reperfusion

open access: yesAsian Pacific Journal of Tropical Biomedicine, 2015
Brain ischemia and reperfusion is the leading cause of serious and long-range disability in the world. Clinically significant changes in central nervous system function are observed following brain ischemia and reperfusion.
Nahid Jivad, Zahra Rabiei
doaj   +1 more source

Ischemic postconditioning protects against ischemic brain injury by up-regulation of acid-sensing ion channel 2a

open access: yesNeural Regeneration Research, 2016
Ischemic postconditioning renders brain tissue tolerant to brain ischemia, thereby alleviating ischemic brain injury. However, the exact mechanism of action is still unclear.
Wang-sheng Duanmu   +5 more
doaj   +1 more source

Cortical organotypic slice cultures as a tool to analyze the neurovascular unit in hypoxia/ischemia and hypothermia-induced neuroprotection [PDF]

open access: yes, 2013
Neurons and glial cells of the central nervous system (CNS) communicate and work together to function and execute an array of complex tasks. In addition to them a third cell type which also works to keep the brain alive are the cerebral endothelial cells
Chip, Sophorn
core   +1 more source

Traumatic brain ischemia during neuro intensive care: myth rather than fact [PDF]

open access: yesArquivos de Neuro-Psiquiatria, 2001
In non-missile severe acute brain trauma, brain ischemia was a frequent finding in cadavers. Studies during neuro intensive care, however, have failed to disclose brain ischemia under most circumstances, except when cerebral hemodynamic and metabolic ...
Julio Cruz
doaj   +1 more source

Hyperglycemia and Focal Brain Ischemia [PDF]

open access: yesJournal of Cerebral Blood Flow & Metabolism, 1999
The influence of hyperglycemic ischemia on tissue damage and cerebral blood flow was studied in rats subjected to short-lasting transient middle cerebral artery (MCA) occlusion. Rats were made hyperglycemic by intravenous infusion of glucose to a blood glucose level of about 20 mmol/L, and MCA occlusion was performed with the ...
L, Gisselsson, M L, Smith, B K, Siesjö
openaire   +2 more sources

Hypoxia–ischemia in the immature brain [PDF]

open access: yesJournal of Experimental Biology, 2004
SUMMARYThe immature brain has long been considered to be resistant to the damaging effects of hypoxia and hypoxia–ischemia (H/I). However, it is now appreciated that there are specific periods of increased vulnerability, which relate to the developmental stage at the time of the insult.
Susan J, Vannucci, Henrik, Hagberg
openaire   +2 more sources

Inflammatory Responses in Brain Ischemia [PDF]

open access: yesCurrent Medicinal Chemistry, 2015
Brain infarction causes tissue death by ischemia due to occlusion of the cerebral vessels and recent work has shown that post stroke inflammation contributes significantly to the development of ischemic pathology. Because secondary damage by brain inflammation may have a longer therapeutic time window compared to the rescue of primary damage following ...
Kawabori, Masahito, Yenari, Midori A
openaire   +4 more sources

Neutralizing anti-interleukin-1β antibodies modulate fetal blood–brain barrier function after ischemia

open access: yesNeurobiology of Disease, 2015
We have previously shown that increases in blood–brain barrier permeability represent an important component of ischemia–reperfusion related brain injury in the fetus.
Xiaodi Chen   +12 more
doaj   +1 more source

[18F]DPA-714: Direct Comparison with [11C]PK11195 in a Model of Cerebral Ischemia in Rats [PDF]

open access: yes, 2013
Purpose: Neuroinflammation is involved in several brain disorders and can be monitored through expression of the translocator protein 18 kDa (TSPO) on activated microglia.
Banister, S D   +60 more
core   +1 more source

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