Results 141 to 150 of about 62,354 (276)
Clinical Cardiology, Volume 49, Issue 1, January 2026.Serum CTRP12 levels are significantly lower in CAD patients, and significant correlate to disease severity and dynamic changes after PCI. They may serve as a potential biomarker for diagnosing CAD and predicting ISR occurrence. ABSTRACT Background C1q/tumor necrosis factor‐related protein 12 (CTRP12) plays a protective role in coronary artery disease ...
Botao Zhao, Yuchao Shen
wiley +1 more source
Mice lacking C1q or C3 show accelerated rejection of minor H disparate skin grafts and resistance to induction of tolerance [PDF]
, 2010 Paramita Baruah +10 more
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Glia, Volume 74, Issue 1, January 2026.SCZ astrocytes display an increased pro‐inflammatory profile upon TNF‐α stimulation. Reactive SCZ astrocytes induce microglial‐like cells to assume a dystrophic phenotype. Reactive SCZ astrocytes limit iMGs' synaptoneurosome uptake and migration. ABSTRACT Neuroinflammation, particularly astrocyte reactivity, is increasingly linked to schizophrenia (SCZ)
Pablo L. Cardozo +18 more
wiley +1 more source
Microglial VRK2 Regulates Astrocytic GABA Synthesis and Tonic Inhibition in the Thalamus
Glia, Volume 74, Issue 1, January 2026.In Vrk2‐deficient mice, tonic GABA inhibition is reduced in the mediodorsal thalamus due to impaired astrocytic GABA synthesis. Microglial VRK2 regulates this process via TNF‐α signaling, revealing a glial–glial mechanism of thalamic inhibition.
Dongsu Lee +10 more
wiley +1 more source
C1q nephropathy- unity in diversity.
Journal of renal injury prevention, 2013 Implication for health policy/practice/research/medical educationC1q nephropathy refers to a disorder in which C1q deposits are seen in mesangium on immunofluorescence microscopy and mesangial electron dense deposits on electron microscopy. The diagnosis of C1q nephropathy is based on demonstration of intense C1q (dominant or co-dominant) positivity ...
openaire +2 more sources
Immunity, Inflammation and Disease, Volume 14, Issue 1, January 2026.All in all, silencing MAFG inhibited the activation of A1 astrocyte and neuroinflammation through regulating CRYAB methylation. ABSTRACT Purpose To investigate the effects of MAF bZIP transcription factor G (MAFG) on the transformation of A1/A2 reactive astrocytes and the production of inflammatory factors after spinal cord injury (SCI). Methods An SCI
Xuefei Li +5 more
wiley +1 more source
The endothelial deprotection hypothesis for lupus pathogenesis: the dual role of C1q as a mediator of clearance and regulator of endothelial permeability [PDF]
, 2015 József Prechl, László Czirják
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Organ Transplantation: Current Status, Challenges, and Future Prospects
MedComm, Volume 7, Issue 1, January 2026.Organ transplantation success relies on precise modulation of the alloimmune response across distinct temporal phases of rejection. Hyperacute rejection results from preformed antibodies activating complement and causing thrombosis, while acute rejection involves T cell‐ and antibody‐mediated mechanisms that drive endothelial activation and ...
Xinqiang Li, Ruidong Ding, Jinzhen Cai
wiley +1 more source