Results 151 to 160 of about 17,322 (203)

Complement in acute kidney injury: a convergent pathogenic pathway in multifactorial renal damage. [PDF]

Front Immunol
Yi D, Yang R, Guo Y, Zhou H, Luan J.
europepmc   +1 more source

The Role of Complement Anaphylatoxins (C3a and C5a) in Non-small Cell Lung Cancer: A Systematic Review. [PDF]

Cureus
Skarentzos K, Karagialani DK, Mitros V, Economopoulou P, Foukas P.   +4 more
europepmc   +1 more source

Complement system in cancer: friend or foe of immunotherapy. [PDF]

J Immunother Cancer
Shah HK, Prem S, Wu X, Liszewski MK, Atkinson JP, Singh AK, Kapoor V.   +6 more
europepmc   +1 more source

Activation of the C3a-C3aReceptor-axis is associated with endothelial dysfunction and glycocalyx damage in ST-elevation myocardial infarction. [PDF]

Basic Res Cardiol
Vahldieck C, Löning S, Hamacher C, Fels B, Svensson T, Rudzewski B, Weil J, Kusche K.   +7 more
europepmc   +1 more source

Monoclonal antibody to C5a receptor

, 1994
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Human C5a and C5a analogs as probes of the neutrophil C5a receptor

Molecular Immunology, 1980
Abstract Quantitative assessment of the human neutrophil chemotactic response and immunologic evidence demonstrate that human C5a and/or its physiological equivalent, C5a des Arg' serves as the predominant chemotactic factor in complement activated serum.
D E, Chenoweth, T E, Hugli
openaire   +2 more sources

Ligands for C3a and C5a Receptors [PDF]

, 2023
Prolonged activation of the complement system of blood proteins in response to infection or tissue injury can lead to inflammatory diseases. This thesis investigates the potency, selectivity and structures of reported and rationally designed new ligands for human complement receptors C3aR and C5aR, towards development of new therapeutic agents. Chapter
openaire   +1 more source

Mapping the Ligand-Binding Site on the C5a Receptor:  Arginine⁷⁴ of C5a Contacts Aspartate²⁸² of the C5a Receptor

Biochemistry, 2001
The interaction between the anaphylatoxin C5a and its receptor involves two distinct sites. One site is formed by acidic residues at the receptor N-terminus and contributes to only ligand binding. The second site, responsible for activation, is less well defined.
Cain, S. A.; id_orcid 0000-0001-8417-1058, Coughlan, T., Monk, P. N.   +2 more
openaire   +2 more sources

Role of C5a and C5a Receptor in Sepsis

2006
In experimental sepsis (after cecal ligation and puncture, CLP) there is convincing evidence of complement activation, as there is in human sepsis. In addition, in sepsis involving rodents and humans, blood neutrophils have lost innate immune functions (chemotaxis, phagocytosis, the ability to produce H2O2).
F. S. Zetoune, P. A. Ward, M. S. Huber-Lang   +2 more
openaire   +1 more source