Claudin-4 Overexpression Predicts Poor Survival and Platinum Resistance in Epithelial Ovarian Cancer: A Potential Biomarker for Clinical Decision-Making. [PDF]
Kutlu Ö +6 more
europepmc +1 more source
EHD4 and ASAP2 are critical negative regulators of the claudin‐5‐based endothelial barrier
Cell‐surface CLDN‐5 protein levels can be evaluated using a probe that can bind to the extracellular domains of CLDN‐5. A probe derived from Clostridium perfringens enterotoxin allows us to isolate cells with high CLDN‐5 protein levels from a knockout cell library.
Yosuke Hashimoto +8 more
wiley +1 more source
<i>Listeria monocytogenes</i> invasion in goat brain tissues: mechanisms of blood-brain barrier disruption and regulation of apoptosis and autophagy. [PDF]
Hu Y +10 more
europepmc +1 more source
Molecular pathology of phyllodes tumours of the breast—much more than MED12
Phyllodes tumours can develop from fibroadenomas bearing MED12 variants by the development of pTERT alterations (“MED12 pathway”) or de novo (“MED12 independent pathway”). Grade progression is associated with increasing genetic complexity including cancer driver gene aberrations. Molecular alterations can be useful in assigning grade and distinguishing
Jia‐Min B Pang +3 more
wiley +1 more source
Claudins proteins in brain tumors: expression patterns and therapeutic target. [PDF]
Romanowicz A +2 more
europepmc +1 more source
Loss of Claudin‐18 (Cldn18) expression is a frequent and specific feature of non‐mucinous lung adenocarcinoma (NM‐LUAD). Cldn18 immunohistochemistry may serve as an ancillary tool for distinguishing malignant from non‐neoplastic alveolar epithelium, as well as for differentiating NM‐LUADs with mucin production from M‐LUADs, particularly in small or ...
Stefano Lucà +18 more
wiley +1 more source
[Research progress in diagnosis and treatment of non-tumorous salivary gland diseases]. [PDF]
Cong X, Su J, Wu L, Ding C, Li W, Yu G.
europepmc +1 more source
Inflammatory and Immunological Basis of Periodontal Diseases
The periodontal lesion emerges as an evolving immunological battlefield, where host–microbiome interactions, dysregulated immune responses, fragile resolution mechanisms, and inflammophilic dysbiosis converge to shift the balance from homeostasis to unrestrained tissue destruction.
Giacomo Baima +3 more
wiley +1 more source
Early intestinal barrier changes in A53T transgenic Parkinson's disease mice. [PDF]
Han MN +5 more
europepmc +1 more source

