Results 111 to 120 of about 18,739,280 (231)
Two cases of cutaneous‐type pemphigus vulgaris and a case of pemphigus foliaceus without mucosal involvement despite high anti‐desmoglein 3 autoantibody levels [PDF]
Akari Terada +3 more
openalex +1 more source
OBJECTIVES/SPECIFIC AIMS: Pemphigus vulgaris (PV) is a potentially fatal blistering disease caused by autoantibodies to the keratinocyte adhesion protein desmoglein 3.
Nina Ran +3 more
doaj +1 more source
Non-pathogenic Anti-Desmoglein 3 IgG Autoantibodies in Fogo Selvagem [PDF]
The endemic form of pemphigus foliaceus, fogo selvagem, is caused by IgG autoantibodies directed against desmoglein 1 (Dsg1). Hilario-Vargas and his colleagues describe a high prevalence of IgG autoantibodies against Dsg3, the target antigen of pemphigus vulgaris, in a Brazilian population where fogo selvagem is endemic, although those patients do not ...
openaire +2 more sources
A Case of Palmoplantar Vesiculobullous Lesions
JEADV Clinical Practice, Volume 5, Issue 1, Page 315-318, March 2026.
Ali Abid +3 more
wiley +1 more source
Polymorphic Mucocutaneous Eruption and Septic Shock in a 75‐Year‐Old Man
JEADV Clinical Practice, Volume 5, Issue 1, Page 323-325, March 2026.
Sweta Subhadarshani +2 more
wiley +1 more source
T Cell Recognition of Desmoglein 3 Peptides in Patients with Pemphigus Vulgaris and Healthy Individuals [PDF]
Christian Veldman +6 more
openalex +1 more source
Pemphigus vulgaris and pemphigus foliaceus are cutaneous autoimmune diseases characterized by intraepithelial blisters and autoantibodies to desmosomal glycoproteins. The antigens recognized by pemphigus vulgaris and pemphigus foliaceus autoantibodies are desmoglein-3 (Dsg3) and desmoglein-1 (Dsg1), respectively.
Lin, Mong-Shang +5 more
openaire +2 more sources
Pemphigus vulgaris (PV) is characterized by IgG1 and IgG4 autoantibodies to desmoglein (Dsg) 3, causing suprabasal blistering of skin and mucous membranes.
Agnes Lo +7 more
semanticscholar +1 more source
Comparative Transcriptome Analysis Identifies Desmoglein-3 as a Potential Oncogene in Oral Cancer Cells. [PDF]
Wan H, Teh MT, Mastroianni G, Ahmad US.
europepmc +1 more source

