Results 251 to 260 of about 93,068 (305)

Photobiomodulation restores blood–brain barrier integrity after hypoxia via endothelial von Willebrand factor modulation in a humanised tricellular transwell model

open access: yesThe Journal of Physiology, EarlyView.
Abstract figure legend Photobiomodulation restores blood–brain barrier (BBB) integrity following hypoxic stress. A multicellular human BBB model comprising brain microvascular endothelial cells, astrocytes and pericytes was established in a transwell system and subjected to acute hypoxia.
Mirriam Domocos   +4 more
wiley   +1 more source

A Quarter Century of EHD Protein Research: From Endosomal Recycling to Ciliopathies

open access: yesTraffic, Volume 27, Issue 3, September 2026.
Human EHD protein subcellular localization. ABSTRACT Eps15 homology domain‐containing proteins comprise a conserved family of membrane‐remodeling ATPases that regulate endocytic trafficking, membrane fission, receptor recycling, primary ciliogenesis and membrane dynamics across eukaryotes. Since the initial identification of EHD1 and its Caenorhabditis
Devin Frisby   +3 more
wiley   +1 more source

Ketogenic Strategies in Neonatal Hypoxic-Ischemic Encephalopathy-The Road to Opening Up: A Scoping Review. [PDF]

open access: yesNeurol Int
Falsaperla R   +7 more
europepmc   +1 more source

Neuroprotective Effects of Furanoditerpenes from <i>Spongia (Spongia) Tubulifera</i> through Cyclophilin D Modulation against Ischemia/Reperfusion Injury in BV2 Microglial Cells. [PDF]

open access: yesACS Chem Neurosci
Castedo N   +8 more
europepmc   +1 more source

Spreading depolarization: A wave that precedes and drives cerebral ischemic cell death-Target for neuroprotection. [PDF]

open access: yesJ Cereb Blood Flow Metab
Pensato U   +6 more
europepmc   +1 more source

Autophagy in hypoxia-ischemia induced brain injury

The Journal of Maternal-Fetal & Neonatal Medicine, 2012
Autophagy is an endogenous tightly regulated process responsible for the degradation of damaged and dysfunctional cellular organelles and protein aggregates. Emerging data indicate a strong and complex interaction among autophagy, apoptosis and necrosis. We studied these interactions in a neonatal model of hypoxia-ischemia (HI).
BALDUINI, WALTER   +2 more
openaire   +5 more sources

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