Results 171 to 180 of about 120,663 (190)
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Metabolic Aspects of Cerebral Hypoxia-Ischemia
1977This communication, which is focused on changes in brain metabolism in hypoxia and ischemia, briefly summarizes results obtained in this and other laboratories during the last 4–5 years. An attempt will be made to describe the characteristic features of hypoxic hypoxia, both at normal and at reduced perfusion pressures, as well as of incomplete and ...
B. K. Siesjö +2 more
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Potential New Therapies for Perinatal Cerebral Hypoxia-Ischemia
Clinics in Perinatology, 1993Recent studies in neonatal animals have shown that hypoxic-ischemic brain damage can be reduced by interventions initiated after the hypoxic-ischemic insult. In this article, the authors focus on potentially new modalities of therapy capable of preventing--or at least reducing--brain damage arising from perinatal cerebral hypoxia-ischemia.
C, Palmer, R C, Vannucci
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Immediate early gene induction after neonatal hypoxia-ischemia
Molecular Brain Research, 1993Immediate early gene (IEG) products, such as FOS and JUN, may partially mediate the long-term transcriptional response of CNS cells to specific changes in their environment. To determine whether IEG products might be involved in the immature brain's response to hypoxia-ischemia (H-I), 7-day-old rat pups were subjected to unilateral common carotid ...
R M, Gubits +4 more
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Progressive Multicystic Encephalopathy: Is There More Than Hypoxia-Ischemia?
Journal of Child Neurology, 2007Progressive multicystic encephalopathy following prenatal or perinatal hypoxia-ischemia is a well-described phenomenon in the literature. The authors report on a term infant with a devastating encephalopathy and severe neuronal dysfunction immediately after delivery without a known antecedent of prenatal or perinatal hypoxia or distress.
Garten, Lars +7 more
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[Molecular factors of cerebral hypoxia-ischemia].
Revista de neurologia, 1996In recent years much has been learned about the cellular and molecular events underlying cerebral hypoxia-ischemia (HI). We review, from a molecular standpoint, the main pathogenetic theories in hypoxic-ischemic cerebral injury, including excitotoxicity, free radical damage, and the role of growth factors, proto-oncogenes and heat shock proteins.
A, Macaya +3 more
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Trans-sodium crocetinate for treating hypoxia/ischemia
Expert Opinion on Investigational Drugs, 2008Trans-sodium crocetinate (TSC) is a novel compound that offers promise as a treatment for conditions caused by hypoxia or ischemia. TSC was originally developed at the University of Virginia for hemorrhagic shock, as part of the battlefield casualty program sponsored by the US Office of Naval Research.
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Perinatal Hypoxia-Ischemia and Brain Injury
Pediatric Research, 2000Edwards, A. D., Azzopardi, D. V.
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[Cell death in neonatal hypoxia-ischemia].
Revista de neurologia, 2000To review the mechanisms of cell death (CD) in the developing brain after hypoxia-ischemia (HI).The possibility of limiting the CD which occurs in serious situations of cerebral HI is based on advances in the understanding of the underlying cellular and molecular mechanisms. Various experimental models offer the opportunity to study these mechanisms in
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[Epileptic signs of neonatal hypoxia-ischemia].
Revista de neurologia, 2000Neonatal convulsions are usually symptomatic and originate mainly from a hypoxic-ischemic (H-I) lesion. The high incidence of crises and their serious sequelas have led us to carry out this retrospective study.We studied 54 histories of neonatal H-I and epileptic crises, analyzing 45 variables and evaluating their diagnostic concordance.In 20 children ...
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