Results 31 to 40 of about 8,158 (292)

Renal experiences of bariatric surgery in patients with type 2 diabetes mellitus [PDF]

open access: yesТерапевтический архив, 2018
The review addresses the questions of the literature devoted to the problem of the influence of bariatric surgery on the course of diabetic nephropathy in patients with diabetes mellitus type 2 after achieving a surgically induced remission.
I I Larina   +3 more
doaj   +1 more source

Nephroprotective Properties of the Glucose-Dependent Insulinotropic Polypeptide (GIP) and Glucagon-like Peptide-1 (GLP-1) Receptor Agonists

open access: yesBiomedicines, 2022
Diabetes mellitus is the leading cause of chronic kidney disease, and about 30–40% of patients with diabetes will develop kidney disease. Incretin hormones have received attention during the past three decades not only as a pharmacotherapy for the ...
Tomislav Bulum
doaj   +1 more source

Update on incretin hormones [PDF]

open access: yesAnnals of the New York Academy of Sciences, 2011
The incretin hormones glucagon‐like‐peptide‐1 (GLP‐1) and glucose‐dependent insulinotropic polypeptide (GIP) are released from the intestine following oral ingestion of nutrients. Incretins promote insulin secretion, while GLP‐1 also inhibits glucagon release and gastric emptying, minimizing postprandial glucose excursions.
Phillips, Liza K., Prins, Johannes B.
openaire   +5 more sources

Incretins today: multiple effects and therapeutic potential [PDF]

open access: yes, 2019
Glucose-dependent insulinotropic peptide (GIP) and glucagon-like peptide-1 (GLP-1) are the incretin hormones initially discovered in the 1960s. GIP and GLP-1 have gained great scientific interest due to their properties in increasing insulin secretion ...
Varvara V. Veretyuk   +2 more
core   +1 more source

Renal effects of glucagon-like peptide receptor agonists in patients with type 1 diabetes mellitus [PDF]

open access: yesТерапевтический архив, 2018
The purpose of our study is to assess the effects of glucagon-like peptide-1 receptor agonists (GLP-1R agonists) on early markers of kidney damage in patients with type 1 diabetes mellitus (DM). Materials and methods.
O K Vikulova   +6 more
doaj   +1 more source

Features of the functional state of the enteropancreatic hormonal system in pregnant women with gestational diabetes mellitus

open access: yesСахарный диабет, 2023
Features of the functional state of the enteropancreatic hormonal system in pregnant women with gestational diabetes mellitus© Fatima O. Ushanova1*, Tatiana Y. Demidova1, Tatiana N. Korotkova21Pirogov Russian National Research Medical University, Moscow,
F. O. Ushanova   +2 more
doaj   +1 more source

timeline incretins

open access: yes, 2014
BMJ OpenData Campaign - Incretins  
Jorge H Ramirez (587826)
core   +1 more source

Glucagon-like peptide-1 analogues: An overview

open access: yesIndian Journal of Endocrinology and Metabolism, 2013
Abnormalities of the incretin axis have been implicated in the pathogenesis of type 2 diabetes mellitus. Glucagon-like peptide-1 (GLP-1) and gastroinhibitory intestinal peptide constitutes >90% of all the incretin function.
Vishal Gupta
doaj   +1 more source

Adipocyte Myoglobin Is a Determinant of Energy Expenditure and a Potential Target to Limit Obesity

open access: yesAdvanced Science, EarlyView.
Myoglobin, known as a muscle oxygen‐carrying protein, is shown to play a key role in fat cells that burn energy. Loss of myoglobin reduces the body's ability to generate heat and increases obesity risk, while restoring it improves metabolism. The study identifies myoglobin as a regulator of fat burning and a potential target to enhance energy ...
Christian Strehlau   +22 more
wiley   +1 more source

GLP-1 and GIP in Type 1 Diabetes Therapy: A Time for Reappraisal?

open access: yesDiabetology
Incretin hormones, such as glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1), are essential regulators of glucose homeostasis, energy balance, and metabolic communication between organs.
Grazia Piras   +6 more
doaj   +1 more source

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