Results 181 to 190 of about 590,806 (400)
Insulin and Insulin-Like Growth Factor I Both Stimulate Metabolism, Growth, and Differentiation in the Postneurula Chick Embryo* [PDF]
M. Marqués Girbau+3 more
openalex +1 more source
Photonic Nanomaterials for Wearable Health Solutions
This review discusses the fundamentals and applications of photonic nanomaterials in wearable health technologies. It covers light‐matter interactions, synthesis, and functionalization strategies, device assembly, and sensing capabilities. Applications include skin patches and contact lenses for diagnostics and therapy. Future perspectives emphasize AI‐
Taewoong Park+3 more
wiley +1 more source
Insulin-like growth factor I sensitization rejuvenates sleep patterns in old mice. [PDF]
Zegarra-Valdivia JA+11 more
europepmc +1 more source
De Novo Design of Integrin α5β1 Modulating Proteins to Enhance Biomaterial Properties
NeoNectins are de novo‐designed miniproteins that selectively bind and stabilize the extended open form of integrin α5β1. When grafted onto biomaterials including hydrogel and titanium, they promote cell attachment and spreading in vitro and promoting tissue integration and bone growth in animal models, demonstrating broad potential for regenerative ...
Xinru Wang+25 more
wiley +1 more source
Combination therapy of insulin-like growth factor I and BTP-2 markedly improves lipopolysaccharide-induced liver injury in mice. [PDF]
Nehme A+10 more
europepmc +1 more source
Total synthesis of insulin-like growth factor I (somatomedin C). [PDF]
C H Li+4 more
openalex +1 more source
Insulin-like growth factor-I is necessary for neural stem cell proliferation and demonstrates distinct actions of epidermal growth factor and fibroblast growth factor-2 [PDF]
Neural stem cells (NSCs), when stimulated with epidermal growth factor (EGF) or fibroblast growth factor-2 (FGF-2), have the capacity to renew, expand, and produce precursors for neurons, astrocytes, and oligodendrocytes.
Aebischer, P.+3 more
core
The artesunate nanoplatform selectively targets ECM CAF, functioning as a GTPase inhibitor through disruption of intracellular serine homeostasis. This metabolic intervention effectively suppresses MAPK cascade activity, which consequently inhibits PTT‐induced CAF to ECM CAF differentiation.
Dongdong Zheng+19 more
wiley +1 more source