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Editor's Note: Protective effects of antidepressant citalopram against abnormal APP processing and amyloid beta-induced mitochondrial dynamics, biogenesis, mitophagy and synaptic toxicities in Alzheimer's disease. [PDF]
Hum Mol Geneteuropepmc +1 more source
SOX2 promotes the progression of hepatocellular carcinoma by targeting FOXJ3 and activating the PI3K/AKT pathway to regulate autophagy and mitophagy. [PDF]
CytojournalZhang Z, Ren W.
europepmc +1 more source
New perspectives on the physiological basis of muscle loss
Experimental Physiology, EarlyView.
Alistair J. Monteyne, Marlou L. Dirks
wiley +1 more source
From Earth to orbit: How to preserve muscle health in space and bed rest
Experimental Physiology, EarlyView.
Antonios Matsakas, Colleen Deane
wiley +1 more source
Nature Reviews Molecular Cell Biology, 2010 Autophagy not only recycles intracellular components to compensate for nutrient deprivation but also selectively eliminates organelles to regulate their number and maintain quality control. Mitophagy, the specific autophagic elimination of mitochondria, has been identified in yeast, mediated by autophagy-related 32 (Atg32), and in mammals during red ...
Richard J Youle +2 more
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Biochimica et Biophysica Acta (BBA) - General Subjects, 2021
Mitochondria play a central role in primary metabolism in plants as well as in heterotrophic eukaryotes. Plants must control the quality and number of mitochondria in response to a changing environment, across cell types and developmental stages. Mitophagy is defined as the degradation of mitochondria by autophagy, an evolutionarily conserved system ...
Sakuya, Nakamura +2 more
openaire +2 more sources
Mitochondria play a central role in primary metabolism in plants as well as in heterotrophic eukaryotes. Plants must control the quality and number of mitochondria in response to a changing environment, across cell types and developmental stages. Mitophagy is defined as the degradation of mitochondria by autophagy, an evolutionarily conserved system ...
Sakuya, Nakamura +2 more
openaire +2 more sources
Mitophagy receptor FUNDC1 regulates mitochondrial dynamics and mitophagy [PDF]
Autophagy, 2016 Mitochondrial fragmentation due to imbalanced fission and fusion of mitochondria is a prerequisite for mitophagy, however, the exact "coupling" of mitochondrial dynamics and mitophagy remains unclear. We have previously identified that FUNDC1 recruits MAP1LC3B/LC3B (LC3) through its LC3-interacting region (LIR) motif to initiate mitophagy in mammalian ...
Ziheng Chen, Zheng Tan, Yanjun Li
exaly +3 more sources
The International Journal of Biochemistry & Cell Biology, 2014
Eukaryotes employ elaborate mitochondrial quality control to maintain the function of the power-generating organelle. Mitochondrial quality control is particularly important for the maintenance of neural and muscular tissues. Mitophagy is specialized version of the autophagy pathway. Mitophagy delivers damaged mitochondria to lysosomes for degradation.
Nobutaka, Hattori +2 more
openaire +2 more sources
Eukaryotes employ elaborate mitochondrial quality control to maintain the function of the power-generating organelle. Mitochondrial quality control is particularly important for the maintenance of neural and muscular tissues. Mitophagy is specialized version of the autophagy pathway. Mitophagy delivers damaged mitochondria to lysosomes for degradation.
Nobutaka, Hattori +2 more
openaire +2 more sources
Biochemistry (Moscow), 2019
Mitochondria play a crucial role in energy production, general cell metabolism, cell signaling, and apoptosis. Mitochondria are also the main source of reactive oxygen species, especially in the case of their dysfunction. Therefore, damaged or even superfluous mitochondria not required for normal cell functioning represent risk factors and should be ...
D V, Mamaev, R A, Zvyagilskaya
openaire +2 more sources
Mitochondria play a crucial role in energy production, general cell metabolism, cell signaling, and apoptosis. Mitochondria are also the main source of reactive oxygen species, especially in the case of their dysfunction. Therefore, damaged or even superfluous mitochondria not required for normal cell functioning represent risk factors and should be ...
D V, Mamaev, R A, Zvyagilskaya
openaire +2 more sources