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Pathology International, Volume 76, Issue 9, September 2026.Histological morphology of an acral fibrochondromyxoid tumor, low power magnification shows the tumor appeared as a clearly defined nodule. The tumor was divided into nodular structures by fibrous septa, chondrocyte‐like tumor cells were embedded within the abundant chondromyxoid stroma, and most cells exhibited a haphazard distribution. ABSTRACT Acral
Liying Zhang +4 more
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Myelin-associated Glycoproteinとニューロパチー
Myelin-associated Glycoproteinとニューロパチーopenaire +3 more sources
Anti-myelin-associated glycoprotein neuropathy
Current Opinion in Neurology, 2006 The anti-myelin-associated glycoprotein (MAG) neuropathy is an antibody-mediated demyelinating neuropathy. The clinical picture is characterized by a distal and symmetric, mostly sensory neuropathy. Monoclonal immunoglobulin M anti-MAG antibodies are uniquely found in this condition and are believed to be pathogenic.
Steck AJ, Stalder AK, Renaud S
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Microheterogeneity of anti-myelin-associated glycoprotein antibodies
Journal of the Neurological Sciences, 2003 Antibodies to the myelin-associated glycoprotein (MAG) are implicated in the pathogenesis of an acquired demyelinating polyneuropathy. We studied IgM affinity to MAG in 18 patients with anti-MAG antibodies. Binding of sera was tested for anti-MAG immunoreactivity in central nervous system (CNS) by ELISA and in CNS and peripheral nervous system (PNS) by
Fluri F, Ferracin F, Erne B, Steck AJ
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A Radioimmunoassay for the Myelin‐Associated Glycoprotein
Journal of Neurochemistry, 1982Abstract: The myelin‐associated glycoprotein (MAG) was purified from rat brain and radioiodinated with Bolton‐Hunter reagent for use in a double‐antibody radioimmunoassay. The conditions of the assay were adjusted to measure between 2 and 30 ng of MAG.
D, Johnson, R H, Quarles, R O, Brady
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Myelination in the absence of myelin-associated glycoprotein
Nature, 1994The hypothesis that myelin-associated glycoprotein (MAG) initiates myelin formation is based in part on observations that MAG has an adhesive role in interactions between oligodendrocytes and neurons. Furthermore, the over- or underexpression of MAG in transfected Schwann cells in vitro leads to accelerated myelination or hypomyelination, respectively.
C, Li +7 more
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Myelin-associated glycoprotein in human retina
Nature, 1984The human retina is unmyelinated, but structural similarities have been noted between Müller cells, the main glial cell type of retina, and oligodendrocytes, the myelin-forming cells of the central nervous system. We now show that antibodies against myelin-associated glycoprotein, a minor component of central and peripheral myelin so far found only in ...
K, Stefansson +6 more
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Myelin-Associated Glycoprotein
1997The myelin-associated glycoprotein (MAG) is quantitatively a minor constituent of the total protein found in myelin isolated from the central (≈1.0%) and peripheral (≈0.1%) nervous systems (Quarles et al. 1973a; Figlewicz et al. 1981). MAG has an apparent molecular weight of 100kD, of which 30% is carbohydrate. Although its precise function is unknown,
Xinghua Yin, Bruce D. Trapp
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The Myelin Associated Glycoprotein In Demyelinating Diseases
1984The myelin associated glycoprotein (MAG) is an integral protein of both central nervous system (CNS) and peripheral nervous system (PNS) myelin. Its molecular weight is approximately 100K Daltons (Quarles, 1979; Figlewicz et al., 1981), and its concentration is 2.7µg/mg protein in adult rat brain and 0.65µg/mg in sciatic nerve (Johnson et al., 1982 ...
N. Latov, E. Nobile-Orazio
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Entrapment in anti myelin-associated glycoprotein neuropathy
Journal of Neurology, 2009Anti-myelin associated glycoprotein (MAG) neuropathy is a chronic disorder in which IgM antibodies react with Schwann cell glycoproteins, including MAG and peripheral myelin protein 22 (PMP22). Nerve conduction studies show features of axon loss and predominantly distal slowing consistent with demyelination.
Catharina G, Faber +3 more
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