Results 151 to 160 of about 6,865 (203)
Salient experiences enhance mundane memories through graded prioritization. [PDF]
Sci AdvLin CL +5 more
europepmc +1 more source
Correction to "Failure to Protect Against Myocardial Ischemia-Reperfusion Injury With Sevoflurane Postcondioning in Old Rats In Vivo". [PDF]
Acta Anaesthesiol Scandeuropepmc +1 more source
Postconditioning the Human Heart [PDF]
Circulation, 2005 Background— In animal models, brief periods of ischemia performed just at the time of reperfusion can reduce infarct size, a phenomenon called postconditioning. In this prospective, randomized, controlled, multicenter study, we investigated whether postconditioning may
Michel Ovize, Gilles Rioufol
exaly +3 more sources
Postconditioning: A Form of “Modified Reperfusion” Protects the Myocardium by Activating the Phosphatidylinositol 3-Kinase-Akt Pathway [PDF]
Circulation Research, 2004 Brief intermittent episodes of ischemia and reperfusion, at the onset of reperfusion after a prolonged period of ischemia, confer cardioprotection, a phenomenon termed "ischemic postconditioning" (Postcond).
Derek Hausenloy, Mihaela M Mocanu
exaly +1 more source
Delayed Postconditioning in the Mouse Heart in Vivo
Circulation, 2011 Background— Reperfusion during acute myocardial infarction remains the best treatment for reducing infarct size. Postconditioning, applied at the onset of reperfusion, reduces myocardial infarction both in animals and humans. The
Stephanie Barrere-Lemaire, Anne Vincent
exaly +2 more sources
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Heart Failure Reviews, 2007
Acute myocardial infarction is the leading cause of morbidity and mortality in industrialized countries. Ischemic postconditioning, that consists of repeated brief episodes of ischemia-reperfusion performed just after reflow following a prolonged ischemic insult, dramatically reduces infarct size in animal models.
Hélène, Thibault +2 more
openaire +2 more sources
Acute myocardial infarction is the leading cause of morbidity and mortality in industrialized countries. Ischemic postconditioning, that consists of repeated brief episodes of ischemia-reperfusion performed just after reflow following a prolonged ischemic insult, dramatically reduces infarct size in animal models.
Hélène, Thibault +2 more
openaire +2 more sources
Mitochondria in Postconditioning
Antioxidants & Redox Signaling, 2011Several signal transduction pathways are activated by cardioprotective stimuli, including ischemic or pharmacological postconditioning. These pathways converge on a common target, the mitochondria, and cardioprotection by postconditioning is associated with preserved mitochondrial function after ischemia/reperfusion.
Böngler, Kerstin +2 more
openaire +3 more sources
Ischemic Postconditioning: Experimental Models and Protocol Algorithms
Basic Research in Cardiology, 2009 Ischemic postconditioning, a simple mechanical maneuver at the onset of reperfusion, reduces infarct size after ischemia/reperfusion. After its first description in 2003 by Zhao et al.
Gerd Heusch, Rainer Schulz
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Long-Term Benefit of Postconditioning [PDF]
Circulation, 2008 Background— We previously demonstrated that ischemic postconditioning decreases creatine kinase release, a surrogate marker for infarct size, in patients with acute myocardial infarction. Our objective was to determine whether ischemic postconditioning could afford (1)
Catherine Sportouch, Hélène Thibault
exaly +3 more sources