Results 241 to 250 of about 304,659 (296)
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Zeitschrift für Rheumatologie, 2011
T-Helfer-Zellen tragen mit der Sekretion von Zytokinen zur Induktion und Aufrechterhaltung rheumatischer Entzundungen bei. Die Untersuchung von Interferon-γ-produzierenden Th1-Zellen, Interleukin-17-produzierenden Th17-Zellen und die vor Kurzem beschriebenen Th1+17-Zellen konnten Einblicke in pathophysiologische Mechanismen rheumatischer Entzundungen ...
H.-D. Chang +2 more
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T-Helfer-Zellen tragen mit der Sekretion von Zytokinen zur Induktion und Aufrechterhaltung rheumatischer Entzundungen bei. Die Untersuchung von Interferon-γ-produzierenden Th1-Zellen, Interleukin-17-produzierenden Th17-Zellen und die vor Kurzem beschriebenen Th1+17-Zellen konnten Einblicke in pathophysiologische Mechanismen rheumatischer Entzundungen ...
H.-D. Chang +2 more
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Current Opinion in Hematology, 2001
After activation, CD4 helper T (Th) cells differentiate into Th1or Th2 effector subsets. These two types of cells produce distinct profiles of cytokines and regulate different immune responses. This review summarizes recent progress on different features of co-stimulatory regulation and chemokine-mediated homing of Th1 and Th2 cells.
C, Dong, R A, Flavell
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After activation, CD4 helper T (Th) cells differentiate into Th1or Th2 effector subsets. These two types of cells produce distinct profiles of cytokines and regulate different immune responses. This review summarizes recent progress on different features of co-stimulatory regulation and chemokine-mediated homing of Th1 and Th2 cells.
C, Dong, R A, Flavell
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Sarcoidosis and Th1 chemokines.
La Clinica terapeutica, 2015Many studies have shown high levels of the chemokine Interferon-γ-inducible protein 10 (IP-10) and its chemokine (C-X-C motif) receptor (CXCR) 3, expressed on Type-1 helper (Th1) lymphocytes, in bronchoalveolar lavage (BAL) and biopsy samples of patients with sarcoidosis.
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Autoreactive effector CD4+ T cells have been associated with the pathogenesis of autoimmune disorders. Early studies implicated the interferon (IFN)‐γ‐producing T helper (Th)1 subset of CD4+ cells as the causal agents in the pathogenesis of autoimmunity. However, further studies have suggested a more complex story. In models thought to be driven by Th1
Rachel Caspi
exaly +3 more sources
2011
???????????? ?????????????????? ???????????????????????? ?????????????????? ?????????????????????? ?????????????????? ?? ???????? ?????????????????? ???????????? ?????? ?????????????????? ???????????? ????????-???????????????????? ?????????? ?????????????????????? ???????????? TNF-??, INF-?? ?? IL-2 ?? ?????????????????? ??????????. ????????????????????
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???????????? ?????????????????? ???????????????????????? ?????????????????? ?????????????????????? ?????????????????? ?? ???????? ?????????????????? ???????????? ?????? ?????????????????? ???????????? ????????-???????????????????? ?????????? ?????????????????????? ???????????? TNF-??, INF-?? ?? IL-2 ?? ?????????????????? ??????????. ????????????????????
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Immunology Today, 1997
Abstract The Th1/Th2 paradigm provides a useful model for understanding the pathogenesis of several diseases, as well as for developing novel immunotherapeutic strategies. Here, Sergio Romagnani examines Th1/Th2 polarization in the context of associated pathophysiological conditions.
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Abstract The Th1/Th2 paradigm provides a useful model for understanding the pathogenesis of several diseases, as well as for developing novel immunotherapeutic strategies. Here, Sergio Romagnani examines Th1/Th2 polarization in the context of associated pathophysiological conditions.
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Th1/Th2 balance in preeclampsia
Journal of Reproductive Immunology, 2003The syndrome of preeclampsia has previously been ascribed to generalized maternal endothelial dysfunction, poor placentation and excessive maternal inflammatory response. Recent reports suggest that preeclampsia is associated with a Th1 predominant profile and may be considered as a failure of the tolerance system allowing the second physiological ...
Masatoshi Sakai
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1996
Markers of TH1 and TH2 lymphocyte subpopulations.
ALMERIGOGNA, FABIO +3 more
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Markers of TH1 and TH2 lymphocyte subpopulations.
ALMERIGOGNA, FABIO +3 more
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Zeitschrift fur Rheumatologie, 2012
T helper cells contribute to the induction and maintenance of rheumatic inflammation through the secretion of cytokines. The analysis of Th1 cells expressing interferon-γ, Th17 cells expressing interleukin-17 and the newly described Th1+17 cells could give insight into the pathophysiological mechanisms of rheumatic diseases.
H-D, Chang, T, Kamradt, H, Schulze-Koops
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T helper cells contribute to the induction and maintenance of rheumatic inflammation through the secretion of cytokines. The analysis of Th1 cells expressing interferon-γ, Th17 cells expressing interleukin-17 and the newly described Th1+17 cells could give insight into the pathophysiological mechanisms of rheumatic diseases.
H-D, Chang, T, Kamradt, H, Schulze-Koops
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Inflammatory Bowel Diseases, 1999
A large body of evidence indicates the existence of functionally polarized CD4+ T-cell responses based on their profile of cytokine secretion. Type 1 T helper (Th1) cells produce interferon-gamma, interleukin (IL)-2, and tumour necrosis factor (TNF)-beta, which activate macrophages and are responsible for cell-mediated immunity and phagocyte-dependent ...
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A large body of evidence indicates the existence of functionally polarized CD4+ T-cell responses based on their profile of cytokine secretion. Type 1 T helper (Th1) cells produce interferon-gamma, interleukin (IL)-2, and tumour necrosis factor (TNF)-beta, which activate macrophages and are responsible for cell-mediated immunity and phagocyte-dependent ...
openaire +2 more sources

