Results 1 to 10 of about 25,619 (108)

Advanced Glycation End-Products (AGEs): Formation, Chemistry, Classification, Receptors, and Diseases Related to AGEs [PDF]

Cells, 2022
Advanced glycation end-products (AGEs) constitute a non-homogenous, chemically diverse group of compounds formed either exogeneously or endogeneously on the course of various pathways in the human body.
Aleksandra Twarda-Clapa, Aleksandra Olczak, Aneta M. Białkowska, Maria Koziołkiewicz   +3 more
doaj   +4 more sources

Advanced Glycation End-Products (AGEs) and Their Soluble Receptor (sRAGE) in Women Suffering from Systemic Lupus Erythematosus (SLE) [PDF]

Cells, 2021
Systemic lupus erythematosus (SLE) is characterized by abnormal action of the immune system and a state of chronic inflammation. The disease can cause life-threatening complications.
Agnieszka Nowak, Brygida Przywara-Chowaniec, Aleksandra Damasiewicz-Bodzek, Dominika Blachut, Ewa Nowalany-Kozielska, Krystyna Tyrpień-Golder   +5 more
doaj   +4 more sources

Study on the Inhibitory Effect of FOs on Advanced Glycation End Products (AGEs) Formation [PDF]

Foods
This study focused on the inhibitory effects of wheat bran feruloyl oligosaccharides (FOs) on the formation of AGEs in three bovine serum albumin (BSA)-based non-enzymatic glycation models, namely BSA-fructose, BSA-methylglyoxal (MGO), and BSA-glyoxal ...
Yongmei Lyu, Haoxiang Wang, Xinying Ye, Zhihan Ge, Wanjie Mao, Zhipeng Cai, Xiaoyang Zhang, Wenlin Sun, Xiaohong Yu   +8 more
doaj   +2 more sources

Oxidative Stress, Advanced Glycation End Products (AGEs), and Neurodegeneration in Alzheimer’s Disease: A Metabolic Perspective [PDF]

Antioxidants
Neurodegenerative diseases such as Alzheimer’s disease (AD) are closely linked to oxidative stress and advanced glycation end products (AGEs), two interrelated processes that exacerbate neuronal damage through mitochondrial dysfunction, protein ...
Virginia Boccardi, Francesca Mancinetti, Patrizia Mecocci   +2 more
doaj   +2 more sources

Lens Autofluorescence Based Advanced Glycation End Products (AGEs) Measurement to Assess Risk of Osteopenia Among Individuals Under the Age of 50 [PDF]

Medical Devices: Evidence and Research, 2022
Shaoyun Li,1,* Yuefei Li,1,* Xiyan Xu,1 Jian Shao,2 Ruifeng Xie,3 Sheng Liu,1 Li Peng,1 Jin Wang,1 Kaixin Zhou,3 Huyi Feng1 1The Fifth People’s Hospital of Chongqing, Chongqing, People’s Republic of China; 2Guangzhou Laboratory, Guangzhou, People’
Li S, Li Y, Xu X, Shao J, Xie R, Liu S, Peng L, Wang J, Zhou K, Feng H   +9 more
doaj   +2 more sources

In Vitro Methodologies to Study the Role of Advanced Glycation End Products (AGEs) in Neurodegeneration. [PDF]

Nutrients, 2022
Chrysanthou M, Miro Estruch I, Rietjens IMCM, Wichers HJ, Hoppenbrouwers T.   +4 more
europepmc   +2 more sources

Advanced Glycation End Products (AGEs) and Chronic Kidney Disease: Does the Modern Diet AGE the Kidney? [PDF]

Nutrients, 2022
Fotheringham AK, Gallo LA, Borg DJ, Forbes JM.   +3 more
europepmc   +2 more sources

Receptor Mediated Effects of Advanced Glycation End Products (AGEs) on Innate and Adaptative Immunity: Relevance for Food Allergy. [PDF]

Nutrients, 2022
Briceno Noriega D, Zenker HE, Croes CA, Ewaz A, Ruinemans-Koerts J, Savelkoul HFJ, van Neerven RJJ, Teodorowicz M.   +7 more
europepmc   +2 more sources

Dietary Intake of Advanced Glycation End Products (AGEs) and Mortality among Individuals with Colorectal Cancer. [PDF]

Nutrients, 2021
Mao Z, Aglago EK, Zhao Z, Schalkwijk C, Jiao L, Freisling H, Weiderpass E, Hughes DJ, Eriksen AK, Tjønneland A, Severi G, Rothwell J, Boutron-Ruault MC, Katzke V, Kaaks R, Schulze MB, Birukov A, Krogh V, Panico S, Tumino R, Ricceri F, Bueno-de-Mesquita HB, Vermeulen RCH, Gram IT, Skeie G, Sandanger TM, Quirós JR, Crous-Bou M, Sánchez MJ, Amiano P, Chirlaque MD, Barricarte Gurrea A, Manjer J, Johansson I, Perez-Cornago A, Jenab M, Fedirko V.   +36 more
europepmc   +2 more sources

Advanced glycation end products regulate the receptor of AGEs epigenetically

Frontiers in Cell and Developmental Biology, 2023
Advanced glycation end-products (AGEs) can boost their receptor of AGE (RAGE) expression through the downstream signaling pathway to facilitate AGE–RAGE interaction. In this regulation process, the primary signaling pathways are NF-κB and STAT3. However,
Xiaoqing Wu, Xiaoqing Wu, Xuanren Shi, Xiaoyong Chen, Zhanhai Yin   +4 more
doaj   +1 more source