Results 121 to 130 of about 15,992 (171)

Detecting Borrelia burgdorferi–Triggered Seronegative Rheumatoid Arthritis Using [68Ga]Ga‐DOTA–Siglec‐9–Positron Emission Tomography/Computed Tomography

ACR Open Rheumatology, Volume 8, Issue 8, August 2026.
Prince Dadson, Heli Ylä‐Outinen, Simona Malaspina, Mikko Koivumäki, Sirpa Jalkanen, Laura Pirilä, Anne Roivainen   +6 more
wiley   +1 more source

Association Between Chikungunya Infection and Rheumatoid Arthritis in Taiwan—A Cross‐Sectional Study

International Journal of Rheumatic Diseases, Volume 29, Issue 8, August 2026.
ABSTRACT Objectives Chikungunya virus (CHIKV) infection might cause chronic chikungunya arthritis resembling rheumatoid arthritis (RA). The purpose of this study is to investigate the potential role of this virus in the pathogenesis of RA in Taiwan, a non‐endemic area. Methods This cross‐sectional study enrolled 98 RA patients and 200 healthy controls.
Chang‐Yi Yen, Chang‐Chi Lin, Ping‐Chang Lin, Ching‐Yi Tsai, Li‐Teh Liu, Chen‐Hsuan Lin, Chun‐Hong Chen, Chia‐Chun Tseng, Hua‐Chen Chan, Cheng‐Chin Wu, Tsan‐Teng Ou, Jih‐Jin Tsai, Jeng‐Hsien Yen   +12 more
wiley   +1 more source

Serum test for secretory component-containing anti-citrullinated protein antibodies as a novel prognostic tool in rheumatoid arthritis at-risk subjects. [PDF]

J Transl Autoimmun
Martinsson K, Åhammar S, Cîrciumaru A, Réthi B, Ziegelasch M, Hensvold A, Kastbom A.   +6 more
europepmc   +1 more source

Neutrophil Extracellular Traps Exacerbate UVB‐Induced Photodamage in HaCaT Cells and Mouse Skin via CCDC25/MAPK Pathway

Aging Cell, Volume 25, Issue 8, August 2026.
Neutrophil extracellular traps (NETs) play a pivotal role in UVB‐induced skin photodamage. Mechanistically, NETs bind to the CCDC25 receptor on keratinocytes and subsequently activate the intracellular p‐JNK/JNK signaling pathway, thereby exacerbating skin photodamage.
Yang Zou, Junzhi Li, Yuting Peng, Rui Hu, Ruolin Li, Aijun Chen   +5 more
wiley   +1 more source

Prevalence of Rheumatoid Factor and Anti-citrullinated Protein Antibodies in Patients With Post-treatment Lyme Disease. [PDF]

Cureus
Miller JB, Rebman A, Yang T, Aucott J.
europepmc   +1 more source

Secretory anti-citrullinated protein antibodies in serum associate with lung involvement in early rheumatoid arthritis. [PDF]

Rheumatology (Oxford), 2020
Roos Ljungberg K, Joshua V, Skogh T, Eklund A, Sköld CM, Karimi R, Nyrén S, Svärd A, Catrina AI, Kastbom A.   +9 more
europepmc   +1 more source

Age‐Associated B Cells: Origins, Regulation, and Tissue‐Specific Pathogenic Contributions in Autoimmune, Metabolic, and Neurological Diseases

Immunological Reviews, Volume 341, Issue 1, August 2026.
ABSTRACT Age‐associated B cells (ABCs) are a distinct B cell population characterized by coexpression of T‐bet and CD11c. First described in aged female mice and autoimmune‐prone strains, ABCs are now recognized to be associated with multiple human autoimmune diseases, including systemic lupus erythematosus, rheumatoid arthritis, and multiple sclerosis,
Kira Rubtsova
wiley   +1 more source

Circulating secretory component-containing anti-citrullinated protein antibodies prior to symptom onset in rheumatoid arthritis. [PDF]

Rheumatology (Oxford)
Martinsson K, Johansson L, Kastbom A, Rantapää-Dahlqvist S.   +3 more
europepmc   +1 more source

Anti‐Modified Protein Antibodies in Rheumatoid Arthritis: Pathogenic, Protective, or Passive Bystander?

Immunological Reviews, Volume 341, Issue 1, August 2026.
ABSTRACT The presence of anti‐modified protein antibodies (AMPA) is a hallmark of rheumatoid arthritis (RA). AMPA recognize post‐translationally modified proteins and are cross‐reactive. AMPA include anti‐citrullinated protein antibodies (ACPA), anti‐carbamylated protein antibodies (anti‐CarP), and anti‐acetylated protein antibodies (AAPA).
Amber‐Sarai F. L. Stalman, Diane van der Woude   +1 more
wiley   +1 more source

Beyond Sequence: Posttranslational Remodeling of Antigens in Autoimmunity

Immunological Reviews, Volume 341, Issue 1, August 2026.
ABSTRACT Autoimmune responses are often attributed to failed tolerance to self‐proteins, yet protein expression alone cannot explain why certain antigens dominate disease, why autoreactivity emerges under stress, or why specific HLA alleles shape risk.
Cynthiya Shrestha, Holly M. Weisfelder, Thanh D. Do   +2 more
wiley   +1 more source