Results 51 to 60 of about 24,234 (207)

MOPS and coxsackievirus B3 stability

open access: yesVirology, 2017
Study of coxsackievirus B3 strain 28 (CVB3/28) stability using MOPS to improve buffering in the experimental medium revealed that MOPS (3-morpholinopropane-1-sulfonic acid) increased CVB3 stability and the effect was concentration dependent. Over the pH range 7.0-7.5, virus stability was affected by both pH and MOPS concentration.
Steven D, Carson   +2 more
openaire   +2 more sources

Autophagy in coxsackievirus-infected neurons [PDF]

open access: yesAutophagy, 2009
Autophagy is a process to engulf aberrant organelles or protein aggregates into double-membrane vesicles for lysosomal breakdown. Autophagy is a protective process against some intracellular bacteria and viruses; however, it is also used for replication by some viruses, such as poliovirus.
Seung-Yong, Yoon   +5 more
openaire   +2 more sources

Primer sets used for coxsackievirus A24 variant.

open access: yes, 2016
Primer sets used for coxsackievirus A24 variant.
Yung-Chang Yen (2967969)   +13 more
core   +1 more source

Pentavalent Sialic Acid Conjugates Block Coxsackievirus A24 Variant and Human Adenovirus Type 37 – Viruses That Cause Highly Contagious Eye Infections [PDF]

open access: yes, 2020
Coxsackievirus A24 variant (CVA24v) and human adenovirus 37 (HAdV-37) are leading causative agents of the severe and highly contagious ocular infections acute hemorrhagic conjunctivitis and epidemic keratoconjunctivitis, respectively.
thilo, stehle   +12 more
core   +2 more sources

Evasins: Biology and Therapeutic Potential of Broad‐Spectrum Chemokine Inhibitors

open access: yesBasic &Clinical Pharmacology &Toxicology, Volume 139, Issue 4, October 2026.
ABSTRACT Ticks are haematophagous parasites that rely on uninterrupted bloodmeals for survival and need an effective strategy to suppress the host inflammatory response. Leukocyte recruitment is a chemokine‐mediated hallmark of inflammation, which ticks counteract via anti‐chemokine proteins in their saliva named evasins.
Emilia L. Bialek   +3 more
wiley   +1 more source

Coxsackievirus and Adenovirus Receptor Cytoplasmic and Transmembrane Domains Are Not Essential for Coxsackievirus and Adenovirus Infection [PDF]

open access: yesJournal of Virology, 1999
ABSTRACTCoxsackievirus and adenovirus receptor (CAR) from which the cytoplasmic domain had been deleted and glycosylphosphatidylinositol (GPI)-anchored CAR lacking both transmembrane and cytoplasmic domains were both capable of facilitating adenovirus 5-mediated gene delivery and infection by coxsackievirus B3.
X, Wang, J M, Bergelson
openaire   +2 more sources

Neutralizing Antibodies against Enteroviruses in Patients with Hand, Foot and Mouth Disease

open access: yesEmerging Infectious Diseases, 2020
Hand, foot and mouth disease (HFMD) is an emerging infection with pandemic potential. Knowledge of neutralizing antibody responses among its pathogens is essential to inform vaccine development and epidemiologic research.
Lam Anh Nguyet   +23 more
doaj   +1 more source

Full-genome analysis of emerging Coxsackievirus B4 genotype D strains associated with herpangina in eastern China. [PDF]

open access: yesMicrob Genom
In 2024 herpangina surveillance in Yuhuan City, China, two Coxsackievirus B4 strains (Z168, Z296) were identified. Full-length genome sequencing revealed that both strains belonged to genotype D that was first documented in eastern China. The two strains
Hu X   +13 more
europepmc   +2 more sources

Oxidative Stress, Post‐Translational Modifications, and Type 1 Diabetes Pathogenesis

open access: yesImmunological Reviews, Volume 343, Issue 1, October 2026.
ABSTRACT Type 1 diabetes (T1D) is an organ‐specific autoimmune disease that destroys insulin‐producing beta‐cells. Insulin is a vital hormone for regulating glycemia that often undergoes post‐translational modifications during processing from pre‐proinsulin to biologically active insulin.
Colleen J. Winstead   +2 more
wiley   +1 more source

Early Treatment of Coxsackievirus B3–Infected Animals With Soluble Coxsackievirus-Adenovirus Receptor Inhibits Development of Chronic Coxsackievirus B3 Cardiomyopathy [PDF]

open access: yesCirculation: Heart Failure, 2019
Background: Coxsackie-B-viruses (CVB) are frequent causes of acute myocarditis and dilated cardiomyopathy, but an effective antiviral therapy is still not available. Previously, we and others have demonstrated that treatment with an engineered sCAR-Fc (soluble coxsackievirus-adenovirus receptor fused to the carboxyl-terminus of
Sandra, Pinkert   +10 more
openaire   +2 more sources

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