Results 61 to 70 of about 22,089 (224)

Immunological Interactions Between Gammadelta T Cells and West Nile Virus in the Infected Host

open access: yesReviews in Medical Virology, Volume 36, Issue 4, July 2026.
ABSTRACT Between mosquitoes and birds, West Nile virus (WNV) is a neurotropic flavivirus, an arthropod‐borne pathogen involved in an enzootic cycle. Additionally, it can infect both people and horses, leading to severe illness. Since 1999, WNV has spread across North and South America, including Mexico and the Caribbean.
E. Cimini   +4 more
wiley   +1 more source

Coxsackievirus B3 Replication and Pathogenesis

open access: yesFuture Microbiology, 2015
Viruses such as coxsackievirus B3 (CVB3) are entirely host cell-dependent parasites. Indeed, they must cleverly exploit various compartments of host cells to complete their life cycle, and consequently launch disease. Evolution has equipped this pico-rna-virus, CVB3, to use different strategies, including CVB3-induced direct damage to host cells ...
Farshid S, Garmaroudi   +7 more
openaire   +2 more sources

The role of inducible nitric oxide synthase in the host response to Coxsackievirus myocarditis

open access: yes, 1998
The host response to Coxsackievirus infection is complex, including T lymphocytes, B lymphocytes, natural killer cells, and macrophages. Although Coxsackievirus infection induces expression of inducible nitric oxide synthase (NOS2; EC 1.14.13.39) in ...
Moncada, Salvador   +17 more
core   +1 more source

Modern Approaches to Enterovirus-Based Oncolytic Immune Virotherapy of Malignant Diseases

open access: yesФармация и фармакология (Пятигорск)
The aim. The review presents the analysis of publications on modern approaches to oncolytic viral immunotherapy of malignant diseases which is predominantly based on usage of enteroviruses.Materials and methods.
E. R. Nemtsova, E. A. Plotnikova
doaj   +1 more source

Coxsackievirus B3, Shandong Province, China, 1990–2010

open access: yesEmerging Infectious Diseases, 2012
To determine the cause of a 2008 outbreak of aseptic meningitis in Shandong Province, China, we analyzed samples from outbreak patients and coxsackievirus B3 samples collected during 1990–2010 surveillance. The cause of the outbreak was coxsackievirus B3,
Zexin Tao   +9 more
doaj   +1 more source

Gingival and Periodontal Diseases and Conditions in Children and Adolescents: Consensus Report

open access: yesJournal of Clinical Periodontology, Volume 53, Issue 7, Page 1068-1099, July 2026.
ABSTRACT Background The objectives of this Focused Workshop were to update the epidemiology, aetiology, risk factors, diagnosis and management of gingival and periodontal diseases and conditions in children and adolescents, and to explore the applicability of the 2018 Classification in children and adolescents.
Iain Chapple   +30 more
wiley   +1 more source

MOPS and coxsackievirus B3 stability

open access: yesVirology, 2017
Study of coxsackievirus B3 strain 28 (CVB3/28) stability using MOPS to improve buffering in the experimental medium revealed that MOPS (3-morpholinopropane-1-sulfonic acid) increased CVB3 stability and the effect was concentration dependent. Over the pH range 7.0-7.5, virus stability was affected by both pH and MOPS concentration.
Steven D, Carson   +2 more
openaire   +2 more sources

Effects of TDP2/VPg Unlinkase Activity on Picornavirus Infections Downstream of Virus Translation

open access: yesViruses, 2020
In this study, we characterized the role of host cell protein tyrosyl-DNA phosphodiesterase 2 (TDP2) activity, also known as VPg unlinkase, in picornavirus infections in a human cell model of infection.
Autumn C. Holmes   +3 more
doaj   +1 more source

RNF213 Is an Interferon‐Stimulated Gene That Targets Influenza A Virus NP and Activates MDA5 to Restrict Infection

open access: yesAdvanced Science, Volume 13, Issue 31, 4 June 2026.
RNF213 is characterized as a dual‐functional antiviral effector. It directly mediates the degradation of the influenza A virus nucleoprotein (NP) while simultaneously activating the MDA5‐mediated innate immune signaling pathway. This coordinated response establishes a powerful host defense system against viral infection. ABSTRACT Influenza A virus (IAV)
Haoning Li   +5 more
wiley   +1 more source

Coxsackievirus and Adenovirus Receptor Cytoplasmic and Transmembrane Domains Are Not Essential for Coxsackievirus and Adenovirus Infection [PDF]

open access: yesJournal of Virology, 1999
ABSTRACTCoxsackievirus and adenovirus receptor (CAR) from which the cytoplasmic domain had been deleted and glycosylphosphatidylinositol (GPI)-anchored CAR lacking both transmembrane and cytoplasmic domains were both capable of facilitating adenovirus 5-mediated gene delivery and infection by coxsackievirus B3.
X, Wang, J M, Bergelson
openaire   +2 more sources

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