Results 101 to 110 of about 97,922 (342)

Eugenia brasiliensis: Analysis of the Chemical Profile and Evaluation of Cytotoxic Potential

Chemistry &Biodiversity, EarlyView.
Leaves of Eugenia brasiliensis were dried, crushed and macerated with polarity increment. The antiporliferative and anti‐migratory potential and the cell cycle were evaluated. 28 compounds were tentatively identified by mass sceptrometry. ABSTRACT This work evaluated the antiproliferative potential of Eugenia brasiliensis leaf extracts against the HeLa
Giovana G. F. V. de Oliveira, Milena F. Longue, Letícia M. R. Pescinelli, Thiago S. Charret, Thalya S. R. Nogueira, Mariana T. M. Pereira, Ivo J. C. Vieira, Lucas S. Abreu, Vinicius D. B. Pascoal, Aislan C. R. F. Pascoal   +9 more
wiley   +1 more source

Demyelination Causes Adult CNS Progenitors to Revert to an Immature State and Express Immune Cues That Support Their Migration

Journal of Neuroscience, 2015
The declining efficiency of myelin regeneration in individuals with multiple sclerosis has stimulated a search for ways by which it might be therapeutically enhanced.
Sarah Moyon, Anne-Laure Dubessy, M. Aigrot, M. Trotter, Jeffrey K. Huang, L. Dauphinot, M. Potier, C. Kerninon, S. Mélik Parsadaniantz, R. Franklin, C. Lubetzki   +10 more
semanticscholar   +1 more source

Delayed Neuropsychiatric Syndrome Due to Unrecognized Carbon Monoxide Toxicity: A Case Report

Annals of the Child Neurology Society, EarlyView.
ABSTRACT Introduction Carbon monoxide (CO) is a leading cause of poison‐related deaths in children and frequently results in nonspecific neurological symptoms and imaging findings. Rarely, pediatric patients develop a delayed neuropsychiatric syndrome (DNS) following a lucid interval.
Alexandria Valdrighi, Greta Peng, Andreas Rauschecker, Mary Karalius   +3 more
wiley   +1 more source

Differential contribution of immune effector mechanisms to cortical demyelination in multiple sclerosis

Acta Neuropathologica, 2017
Cortical demyelination is a widely recognized hallmark of multiple sclerosis (MS) and correlate of disease progression and cognitive decline. The pathomechanisms initiating and driving gray matter damage are only incompletely understood.
Nielsen Lagumersindez-Denis, Claudia Wrzos, M. Mack, A. Winkler, F. van der Meer, M. Reinert, Heiko Hollasch, A. Flach, H. Brühl, E. Cullen, C. Schlumbohm, E. Fuchs, C. Linington, A. Barrantes-Freer, I. Metz, C. Wegner, D. Liebetanz, M. Prinz, W. Brück, C. Stadelmann, S. Nessler   +20 more
semanticscholar   +1 more source

A BIOCHEMICAL AND MORPHOLOGIC STUDY OF MYELINATION AND DEMYELINATION [PDF]

, 1958
Guido Majno, Manfred L. Karnovsky
openalex   +1 more source

Central and peripheral demyelination

Journal of Neurosciences in Rural Practice, 2014
ABSTRACTSeveral conditions cause damage to the inherently normal myelin of central nervous system, perepheral nervous system or both central and perepheral nervous system and hence termed as central demyelinating diseases, perepheral demyelinating diseases and combined central and perepheral demyelinating diseases respectively.
Natasha Singh Gulati, Man Mohan Mehndiratta   +1 more
openaire   +3 more sources

Neuroleukemiosis Masquerading as Drug Toxicity in an Adolescent With Refractory AML

American Journal of Hematology, EarlyView.
Nia Choi, Deborah Schady, Tim Lotze, Jill Ann Jarrell, Alexandra Rodriguez‐Hernandez, Sandra Aziz, Karen K. Moeller, Joanna S. Yi, Suzanne Woodbury, Andrea N. Marcogliese, Zoann E. Dreyer, Eric S. Schafer   +11 more
wiley   +1 more source

Combinatorial expression of glial transcription factors induces Schwann cell‐specific gene expression in mouse embryonic fibroblasts

Developmental Dynamics, EarlyView.
Abstract Background Schwann cells provide peripheral nerve trophic support, myelinate axons, and assist in repair. However, Schwann cell repair capacity is limited by chronic injury, disease, and aging. Schwann cell reprogramming is a cellular conversion strategy that could provide a renewable cell supply to repair injured nerves.
Lauren Belfiore, Anjali Balakrishnan, Yacine Touahri, Dawn Zinyk, Humna Noman, Satoshi Okawa, Jeff Biernaskie, Carol Schuurmans   +7 more
wiley   +1 more source

Blood-brain barrier hyperpermeability precedes demyelination in the cuprizone model

Acta Neuropathologica Communications, 2017
In neuroinflammatory disorders such as multiple sclerosis, the physiological function of the blood-brain barrier (BBB) is perturbed, particularly in demyelinating lesions and supposedly secondary to acute demyelinating pathology.
Stefan A Berghoff, Tim Düking, L. Spieth, J. Winchenbach, S. Stumpf, Nina Gerndt, Kathrin Kusch, T. Ruhwedel, W. Möbius, G. Saher   +9 more
semanticscholar   +1 more source

Microcephaly with Extensive Calcium Deposits and Demyelination [PDF]

, 1954
George A. Jervis
openalex   +1 more source