Results 71 to 80 of about 120,663 (190)

Effect of Differently Polarized Human Macrophages on the SH-SY5Y Cells Damaged by Ischemia and Hypoxia In Vitro

open access: yesJournal of Immunology Research, 2023
Macrophages are the major cells of innate immunity with a wide range of biological effects due to their great plasticity and heterogeneity. Macrophages play a key role in neuroregeneration following nervous tissue injury.
Ivan Mikhailovich Rashchupkin   +5 more
doaj   +1 more source

Long-term deficits in cortical circuit function after asphyxial cardiac arrest and resuscitation in developing rats [PDF]

open access: yes, 2017
Cardiac arrest is a common cause of global hypoxic-ischemic brain injury. Poor neurologic outcome among cardiac arrest survivors results not only from direct cellular injury but also from subsequent long-term dysfunction of neuronal circuits.
Clark, Robert S.B.   +5 more
core   +2 more sources

Photoacoustic oxygenation imaging to identify ischemia/hypoxia injury and necrosis of intestine after acute intussusception: A comparative study with CDFI/CEUS

open access: yesPhotoacoustics
Acute intussusception is a pediatric abdominal emergency that requires immediate diagnosis and treatment. However, accurately identifying bowel necrosis non-invasively remains challenging with conventional sonography.
Hualin Yan   +7 more
doaj   +1 more source

The GluN3A Subunit Exerts a Neuroprotective Effect in Brain Ischemia and the Hypoxia Process

open access: yesASN Neuro, 2013
NMDARs ( N -methyl-d-aspartate receptors) mediate the predominantly excitatory neurotransmission in the CNS (central nervous system). Excessive release of glutamate and overactivation of NMDARs during brain ischemia and the hypoxia process are causally ...
Hui Wang   +5 more
doaj   +1 more source

Preconditioning triggered by carbon monoxide (CO) provides neuronal protection following perinatal hypoxia-ischemia.

open access: yesPLoS ONE, 2012
Perinatal hypoxia-ischemia is a major cause of acute mortality in newborns and cognitive and motor impairments in children. Cerebral hypoxia-ischemia leads to excitotoxicity and necrotic and apoptotic cell death, in which mitochondria play a major role ...
Cláudia S F Queiroga   +5 more
doaj   +1 more source

Apoptosis and inflammation regulation after injury to the developing brain [PDF]

open access: yes, 2015
The brain shows greater plasticity in early life than in maturity, which paradoxically renders the organ more vulnerable to hypoxia-ischemia (HI)- and cranial irradiation (IR)- induced damage.
Han, Wei
core   +1 more source

Strain-Related Differences in Mouse Neonatal Hypoxia-Ischemia [PDF]

open access: yesDevelopmental Neuroscience, 2018
Neonatal hypoxic-ischemic brain injury is commonly studied by means of the Vannucci procedure in mice or rats (unilateral common carotid artery occlusion followed by hypoxia). Previously, we modified the postnatal day 7 (P7) rat procedure for use in mice, and later demonstrated that genetic strain strongly influences the degree of brain injury in the ...
R. Ann Sheldon   +2 more
openaire   +4 more sources

Data on uncoupling protein-3 levels, hypoxia, low flow ischemia, and insulin stimulation in dystrophin-deficient mdx mouse hearts

open access: yesData in Brief, 2018
The data contain body weights, plasma free fatty acids concentrations and cardiac uncoupling protein-3 protein levels for wild-type and mdx mice. The data provide heart rates, left ventricular contractile functions, coronary flow, phosphocreatine ...
Wen Zhang   +4 more
doaj   +1 more source

The Iowa Perinatal Letter, January-February-March 2007, Vol. 28, no.1 [PDF]

open access: yes, 2007
This newsletter from The Department of Public Health about perinatal health care and ...

core  

Treatment temperature and insult severity influence the neuroprotective effects of therapeutic hypothermia [PDF]

open access: yes, 2016
Therapeutic hypothermia (HT) is standard care for moderate and severe neonatal hypoxic-ischaemic encephalopathy (HIE), the leading cause of permanent brain injury in term newborns.
Falck, Mari   +8 more
core   +2 more sources

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