Results 131 to 140 of about 2,944 (198)

Relaxin‐2: Shaping the Proteomic Landscape of Skeletal Muscle Physiology, Glucose Trafficking, and Mitochondrial Function in Rat

The FASEB Journal, Volume 40, Issue 18, 30 September 2026.
Cardioprotective hormone relaxin‐2 showed relevant effects on rat skeletal muscle by altering proteins linked to muscle function, regeneration, differentiation, mitochondrial function, glucose metabolism, and structural integrity and organization. Specifically, relaxin‐2 reduced the expression of 95 proteins, increased 32, and elicited unique proteins ...
Xocas Vázquez‐Abuín, Lucía Gómez‐Cibeira, Javier García‐Seara, Pablo de la Fuente‐López, Carlos Minguito‐Carazo, Susana B. Bravo, Manuel Otero‐Santiago, Oreste Gualillo, José Ramón González‐Juanatey, Francisca Lago, Sandra Feijóo‐Bandín, Alana Aragón‐Herrera   +11 more
wiley   +1 more source

Asynchronous movement of sarcomeres in myocardium under living conditions: role of titin

Frontiers in Physiology
Fuyu Kobirumaki-Shimozawa, Kotaro Oyama, Kotaro Oyama, Tomohiro Nakanishi, Tomohiro Nakanishi, Shin’ichi Ishiwata, Norio Fukuda   +6 more
doaj   +1 more source

PENGARUH ASAM AKROBAT TERHADAP PEMBENTUKAN GEL MIOFIBRIL IKAN MATA BESAR (Selar crumenophthalnus) [effect of ascorbic acid on gel formation of myofibril from big eye scad fish (Selar crumenophthalnus)]

Jurnal Teknologi dan Industri Pangan, 2005
Effect of ascorbic acid on gel formation of myofibril from big eye scad fish (Selar crumenophthalnus)were studied for its development as food ingredient.
Yuli Witono, Mukhamad Fauzi, Wiwik siti Windrati, Achmad Subagio   +3 more
doaj  

Commentary: Nanoscopy reveals the layered organization of the sarcomeric H-zone and I-band complexes

Frontiers in Cell and Developmental Biology, 2020
Nicanor González-Morales, Frieder Schöck   +1 more
doaj   +1 more source

Abstract 4137103: Myofibril Mechanics and Transcriptomic Profiling Confirm Distinct Phenotype of TNNI3- Cardiomyopathy Mouse Model

Introduction: Cardiac troponin I (cTnI, TNNI3 gene) is a highly conserved subunit of the sarcomere that inhibits contraction by preventing actin-myosin interaction. Pathogenic TNNI3 variants can
Fenwick, Axel, Adler, Eric, Gunes, Betul, Cammarato, Anthony, Silver, Elizabeth, Bushway, Paul   +5 more
core   +1 more source

Methods for Analyzing Myofibril Orientation of hIPSC Derived Cardiomyocytes in 3D [PDF]

Human induced pluripotent stem cell derived cardiomyocytes (hiPSC-CMs) are used for studying the function of the heart, as well as cardiac disease and drug response in vitro.
Heino, Jenni
core  

The bound nucleotide of the isolated myofibril [PDF]

Biochemical Journal, 1952
openaire   +2 more sources

The protein components of the isolated myofibril [PDF]

Biochemical Journal, 1953
openaire   +2 more sources

Common Heart Failure With Preserved Ejection Fraction Animal Models Yield Disparate Myofibril Mechanics

Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease
Axel J. Fenwick, Vivek P. Jani, D. Brian Foster, Thomas E. Sharp, Traci T. Goodchild, Kyle LaPenna, Jake E. Doiron, David J. Lefer, Joseph A. Hill, David A. Kass, Anthony Cammarato   +10 more
doaj   +1 more source

Contraction Mechanism of the Myofibril

Denshi kenbikyo, 1954
openaire   +1 more source