Results 61 to 70 of about 6,060 (200)
ABSTRACT Pathophysiological mechanisms of neurological manifestations in COVID‐19 are not fully known yet. In this case‐control study, proteomic analysis was used to characterize cerebrospinal fluid (CSF) samples from COVID‐19 patients with acute neurological manifestations primarily categorized as: isolated refractory headache (n = 12), encephalopathy
Nicole Lardini Freitas +8 more
wiley +1 more source
In nature prion diseases are usually transmitted by extracerebral prion infection, but clinical disease results only after invasion of the central nervous system (CNS).
James F. Striebel +5 more
core +1 more source
Abstract Background Severe COVID‐19 infection requiring hospitalization is associated with oropharyngeal dysphagia, yet trajectories in the inpatient rehabilitation facility (IRF) setting remain undercharacterized. Objective To investigate the prevalence, risk factors, recovery likelihood, and evolution of dietary requirements of patients with ...
Benjamin K. Petrie +3 more
wiley +1 more source
ABSTRACT Emerging evidence links COVID‐19 to the predisposition of Parkinson's disease (PD). However, the relationship between long COVID and prodromal PD‐like features remains unclear, particularly in long COVID participants with dream‐enactment behaviours (DEBs) that may be suggestive of possible REM sleep behaviour disorder.
Siyi Gong +28 more
wiley +1 more source
Dynamic ubiquitination drives herpesvirus neuroinvasion [PDF]
Significance The neuroinvasive herpesviruses cause a wide range of diseases in humans and livestock. These viruses have the unusual ability to routinely enter the mammalian nervous system following infection of mucous membranes.
Huffmaster, Nicholas J. +4 more
openaire +2 more sources
Upon JEV infection, ZNF33B recruits METTL14 to stabilize the METTL3‐METTL14 m6A methyltransferase complex, leading to increased m6A modification of host transcripts, including Trim25 mRNA. ZNF33B selectively binds m6A‐modified sites on Trim25 mRNA and accelerates its decay, resulting in reduced TRIM25 protein abundance.
Jian Du +9 more
wiley +1 more source
Pathogens invade the central nervous system (CNS) and cause infections either through the haematogenous route or via peripheral nerves. Neuroinvasion via peripheral nerves, involving spinal or cranial somatic nerves, is well-established for certain viral
Ong, Kien Chai +5 more
core +2 more sources
Characterization of ovine palatine tonsil innervation: a potential route for scrapie prion ...
MONGARDI FANTAGUZZI C. +6 more
core +1 more source
Pathophysiology and clinical implications of dengue-associated neurological disorders
Dengue virus (DENV), a mosquito-borne Flavivirus, represents a growing global health challenge, particularly in tropical and subtropical regions. The 2009 WHO classification system for dengue categorizes infections into dengue without warning signs ...
Ramtin Naderian +11 more
doaj +1 more source
Expression of the cellular prion protein by mast cells in the human carotid body
Prion diseases are fatal neurologic disorders that can be transmitted by blood transfusion. The route for neuroinvasion following exposure to infected blood is not known.
Gregory D. Sweetland +4 more
doaj +1 more source

