Results 101 to 110 of about 6,782 (204)

SEMA3A mutations identified in Kallmann syndrome patients.

, 2012
SEMA3A mutations identified in Kallmann syndrome patients.
Chantal Metz (141704), Chrystel Leroy (141675), Didier Dewailly (141695), Stéphanie Baron (141677), Gérard Chabrier (141708), Jean-Pierre Hardelin (141712), Catherine Dodé (141716), Paolo Giacobini (141657), Jyoti Parkash (141664), Céline Campagne (141680), Corinne Cruaud (85480), Ksenija Gersak (141701), Corinne Fouveaut (141671), Charlotte Vanacker (141684), Christine Cortet-Rudelli (141698), Pierre Lhuillier (141660), Michel Pugeat (141710), Cécile Espy (141668), Francis Collier (141688), Jacques Young (101833), Alfons García-Piñero (141693), Vincent Meyer (141691), Naresh Kumar Hanchate (141654), Vincent Prevot (141714)   +23 more
core   +1 more source

بررسی ارتباط فاکتورهای حفظ و بازسازی‌کننده پیوندگاه عصبی- عضلانی با فاکتورهای التهابی هیپوکامپ تحت تأثیر تمرین ورزشی شنا در موش‌های مدل آلزایمری [PDF]

مجله دانشگاه علوم پزشکی سبزوار
زمینه و هدف: ورزش یک گزینه غیردارویی امیدوارکننده برای به‌تأخیرانداختن بالقوه شروع یا کندکردن پیشرفت آلزایمر است از این‌رو این پژوهش درصدد یافتن مکانیسم احتمالی ارتباط عضله و هیپوکمپ تحت تأثیر تمرین ورزشی شنا در رت‌های مبتلا به آلزایمر است. مواد و روش­ها:
محمد سلیمانی فارسانی, محمد فتحی, زهرا همتی, زینب گرگین   +3 more
doaj   +1 more source

Semaphorin 3A (SEMA3A) [PDF]

Science-Business eXchange, 2011
openaire   +3 more sources

Evaluation of Morphological Changes in Muscle Fibers and Neuronal Terminals of Motor Neurons in Male Rats from Childhood to Old Age

Majallah-i Dānishgāh-i ’Ulūm-i Pizishkī-i Shahīd Ṣadūqī Yazd, 2022
Introduction: The aim of this study was to investigate the effect of time and age on morphological changes in male rats. Methods: For conducting this study, 15 male Wistar rats in three age groups of 2 weeks (150-100 g), 6 weeks (250-220 g) and 96 weeks (
Mohammad Mahdi Bagherpour Tabalvandani, Mohammad Reza Fadaei Chafy, Alireza Elmieh   +2 more
doaj  

ATI markers are reduced in Sema3A^-/- mice.

, 2013
A. Reduced immunostaining for the type I alveolar epithelial cell marker, T1-alpha (red), is seen in lung from a Sema3A-/- mouse at E17.5 (right) as compared with a Sema3A+/+ littermate control (left).
Michael J. Delannoy (339732), Patrice M. Becker (339730), Tracy S. Tran (339731), Chaoxia He (339733), John M. Shannon (339734), Sharon McGrath-Morrow (216731)   +5 more
core   +1 more source

Loss of inhibitory semaphorin 3A (SEMA3A) autocrine loops in bone marrow endothelial cells of patients with multiple myeloma

, 2006
Vascular endothelial growth factor165 (VEGF165) and semaphorin3A (SEMA3A) elicit pro- and anti-angiogenic signals respectively in endothelial cells (EC) by binding to their receptors VEGFR-2, neuropilin-1 (Nrp- 1), and plexin-A1.
Cirulli T, Bussolino F, RIBATTI, Domenico, Vacca A, Di Pietro G, Merchionne F, Piaggio G, VACCA, Angelo, GUIDOLIN, DIEGO, Serini G, Dammacco F., Scavelli C, Bacigalupo A, GUIDOLIN D, Ribatti D   +14 more
core   +1 more source

Semaphorin 3A Increases in the Plasma of Women with Diminished Ovarian Reserve Who Respond Better to Controlled Ovarian Stimulation

Life
Semaphorin 3A (SEMA3A) plays a crucial role in the development, differentiation, and plasticity of specific types of neurons that secrete Gonadotropin-Releasing Hormone (GnRH) and regulates the acquisition and maintenance of reproductive competence in ...
Michela Palese, Gabriella Ferretti, Giuseppe Perruolo, Sara Serafini, Rossana Sirabella, Vincenzo Marrone, Martina De Rosa, Laura Sarno, Ida Strina, Carmela Matrone, Maurizio Guida   +10 more
doaj   +1 more source

Elimination of aberrant DRG circuitries in Sema3A mutant mice leads to extensive neuronal deficits.

, 2013
Axon guidance molecules determine the pattern of neuronal circuits. Accuracy of the process is ensured by unknown mechanisms that correct early guidance errors.
Sahar Sweetat, Ayal Ben-Zvi, Oded Behar
core   +1 more source

Semaphorin 3A (SEMA3A) [PDF]

Science-Business eXchange, 2012
openaire   +1 more source

Expression of a mutant SEMA3A variant (K108N-SEMA3A) does not significantly alter the sprouting capacity at non-plastic NMJs after BotoxA-induced muscle paralysis in male mice.

, 2017
Neuromuscular junctions in TypeIIb and TypeI/IIa regions of wild-type (WT), K108N-SEMA3A heterozygote (N/-) and K108N-SEMA3A homotzygote (N/N) mouse gastrocnemic muscle were scored as normal or displaying sprouts 14 days after administration of BotoxA ...
Joost Verhaagen (55754), Fred De Winter (3684880), Barbara Hobo (533803), Elizabeth B. Moloney (3684877)   +3 more
core   +1 more source