Results 61 to 70 of about 33,694 (186)
iMeta, Volume 5, Issue 1, February 2026.Research on the mechanism of OPR restoring intestinal epithelial cell (IEC) structure and function in inflammatory bowel disease (IBD) treatment. (A) Schematic diagram of OPR structure and the rate of GP release under different conditions. (B) Schematic diagram of OPR targeting IBD colonic lesions through charge interaction.
Wensheng Chen +7 more
wiley +1 more source
Clostridium BotulinumTypes A and B Toxin-Antitoxin Reactions [PDF]
Journal of Hygiene, 1955 SummaryC. botulinumtypes A and B toxins required larger amounts of antitoxin for neutralization when fractions of the test dose of toxin were used than would be expected.The degree of this dilution effect, i.e. the dilution ratio, varied with different sera.
openaire +2 more sources
Toxigenic and non-toxigenic strains of Clostridium botulinum type E : A. Differentiation by immunodiffusion : B. Attempts to induce toxigenicity by mutagenic agents [PDF]
In this study of toxigenic and non-toxigenic strains of Clostridium botulinum type E, attempts were made to induce toxin production in non-toxigenic cultures by exposure to mutagenic agents and to differentiate the organisms by immunodiffusion.
Balstraz, Edwina Gene
core +5 more sources
Type C botulism outbreak in free-ranging waterfowl in Goiás
Pesquisa Veterinária Brasileira, 2022 : Botulism is generally a fatal disease caused by ingestion of neurotoxins produced by Clostridium botulinum. The present study describes the epidemiological, clinical, and laboratory aspects of a type C botulism outbreak in free-living aquatic birds ...
Andressa S. Martins +7 more
doaj +1 more source
Botulinum toxin-A for the treatment of overactive bladder: UK contributions [PDF]
, 2013 Background: Botulinum toxin-A (BoNT/A) is now established second-line management for refractory overactivebladder (OAB) and recognised in many incontinence guidelines and pathways.
Fowler, CJ +13 more
core +1 more source
MicrobiologyOpen, Volume 14, Issue 6, December 2025.The comparative genomic analysis provides information about how C. tertium HGMC01 strain cause disease through mucin glycan degradation, colonization, multidrug resistance, and modulation of immune responses. Moreover, this analysis data suggest that this strain interacts with host cells by degrading mucin in the gut, potentially through recognition or
Seonghun Kim +2 more
wiley +1 more source
In-Vivo and In-Vitro Studies of the Effect of Specific Antitoxin on the Phagocytosis of Cells of Clostridium Botulinum Type A [PDF]
Journal of Medical Microbiology, 1973 Summary The conversion of heat-resistant spores of Clostridium botulinum type A into germinated heat-sensitive cells in the peritoneal cavity of mice has been observed. This in-vivo germination of spores was depressed in mice passively immunised with type-A botulinal antitoxin.
J B, Suzuki, N, Grecz
openaire +2 more sources
A Targeted Therapeutic Rescues Botulinum Toxin-A Poisoned Neurons [PDF]
, 2008 Botulinum neurotoxin (BoNT), a Category A biothreat agent, is the most potent poison known to mankind. Currently no antidote is available to rescue poisoned synapses.
Radharaman Ray +6 more
core
Metabolic Origin, Role and Fate of the Denaturant Guanidine
Microbial Biotechnology, Volume 18, Issue 11, November 2025.The origin of metabolic guanidine is largely a mystery. We suggest it is created when guanine‐containing nucleotides are oxidised by molecular oxygen instead of being broken down into urea as purines normally would. Guanidine may act as a signal to help cells control the level of reactive oxygen species.
Antoine Danchin +3 more
wiley +1 more source
A severe case of iatrogenic botulism associated with COVID-19 infection
HeliyonBackground: The botulinum toxin is an extremely potent substance that impacts the nervous system. There has been a rise in cases of medical poisoning associated with it, particularly in the field of plastic and aesthetic procedures, in recent years. Case
Qian Ma +6 more
doaj +1 more source