Results 31 to 40 of about 217,483 (203)

Recurrent C3 Glomerulonephritis along with BK-Virus-Associated Nephropathy after Kidney Transplantation: A Case Report. [PDF]

open access: yesMedicina (Kaunas), 2023
C3 glomerulonephritis (C3GN) is a rare cause of end-stage kidney disease and frequently recurrent in allografts following kidney transplantation (KT). Herein, we describe the case of a kidney transplant recipient who developed recurrent C3GN along with ...
Lim JH   +16 more
europepmc   +2 more sources

WCN24-386 C3 GLOMERULONEPHRITIS AND C3-DOMINANT INFECTION RELATED GLOMERULONEPHRITIS - ARE THEY SIMILAR?

open access: yesKidney International Reports
Mythri Shankar   +4 more
doaj   +2 more sources

C3 glomerulonephritis associated with monoclonal gammopathy of renal significance: a diagnostic and therapeutic challenge. [PDF]

open access: yesJ Bras Nefrol
C3 glomerulopathy represents a heterogeneous group of glomerulopathies characterized by hyperactivity of the alternative complement pathway. Although the pathophysiology is incompletely understood, an association between this disease and monoclonal ...
Beirão B   +5 more
europepmc   +3 more sources

Effectiveness of mycophenolate mofetil in C3 glomerulonephritis. [PDF]

open access: yesKidney International, 2015
C3 glomerulonephritis is a clinicopathologic entity defined by the presence of isolated or dominant deposits of C3 on immunofluorescence. To explore the effect of immunosuppression on C3 glomerulonephritis, we studied a series of 60 patients in whom a complete registry of treatments was available over a median follow-up of 47 months.
C. Rabasco   +30 more
semanticscholar   +4 more sources

C3 Glomerulonephritis Associated with Monoclonal Gammopathy. [PDF]

open access: yesIndian J Nephrol
C3 glomerulonephritis (C3GN) is rare. It is a proliferative glomerulonephritis resulting from glomerular deposition of complement factors due to dysregulation of the alternative pathway of complement. The association between monoclonal protein production and development of C3GN was described.
Piedade AD   +4 more
europepmc   +3 more sources

Cat-Scratch Disease Masquerading as C3 Glomerulonephritis. [PDF]

open access: yesKidney Int Rep, 2020
Sutu B   +6 more
europepmc   +5 more sources

C3 glomerulonephritis and CFHR5 nephropathy [PDF]

open access: yesNephrology Dialysis Transplantation, 2012
Complement is an important aspect of defence against infection and its activation and regulation are finely balanced. Disordered complement regulation can lead to C3 glomerulonephritis (C3GN), which is characterized by complement (but not immunoglobulin) deposition in the glomerulus of the kidney. Although only recently recognized as a clinical entity,
Daniel P, Gale, Patrick H, Maxwell
openaire   +2 more sources

Eculizumab and recurrent C3 glomerulonephritis. [PDF]

open access: yesPediatr Nephrol, 2013
Hyperactivity of the alternative complement pathway is the principle defect in C3 glomerulopathies (C3G). Eculizumab, a monoclonal antibody that binds C5 to prevent formation of the membrane attack complex, has been shown to be beneficial in some patients with this disease.In this open-label, proof-of-concept efficacy-and-safety study, a patient with ...
Gurkan S   +5 more
europepmc   +4 more sources

Heterozygous laminin β2 mutation in C3 glomerulopathy

open access: yesSaudi Journal of Kidney Diseases and Transplantation, 2021
C3 glomerulopathy is usually seen with the presence of C3 nephritic factor, homozygous or heterozygous mutations in the regulatory complement proteins factor H, factor I, or C3. We describe the presence of heterozygous laminin β2 mutation in a patient of
Manish R Balwani   +3 more
doaj   +1 more source

A novel mutation in complement 2 accompanied by susceptibility variants in C3 glomerulonephritis: A case study

open access: yesNefrología (English Edition), 2019
Background: C3 glomerulonephritis is a rare, chronic disease characterized by C3c-dominant staining on renal biopsy and is caused by inherited or acquired alternative complement pathway dysregulation.
Sha-sha Han   +5 more
doaj   +3 more sources

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