Results 41 to 50 of about 88,420 (193)

Genetic distance between mammalian CTLA4, B7.1/CD80 and B7.2/CD86.

open access: yes, 2016
The percentage of amino acid identities between pairwise comparisons are shown as a matrix for CTLA4 (A), CD80 (B) and CD86 (C). The distribution of pairwise identities was calculated in segments of 4% (D) and illustrates higher amino acid conservation ...
Wiebke Baars (3170967)   +15 more
core   +1 more source

Nucleic acid encoding feline CD86

open access: yes, 1999
The present invention provides isolated and purified DNA encoding feline CD80 (B7-1) ligand, feline CD86 (B7-2) ligand, feline CD28 receptor, or feline CTLA-4 (CD152) receptor, as well as vectors comprising nucleic acid encoding feline CD80, feline CD86,
Choi, Insoo   +3 more
core   +1 more source

Supplementary figure 1. CD68+CD86-/low M0-macrophage and CD68+CD86+ M1-macrophage phenotypes.

open access: yes, 2022
Representative flow cytometry gating of live cells for the M0 macrophages (negative ctrl) in (A) as well as forward (FSC) and side scatter (SSC) intensity plots to gating Singlet in (B) and morphological analysis in (C). Evaluation of M0 (left) and M1 (right) cell phenotypes (D-G) was determined by flow cytometry as percentage for the expression of the
Gori, Manuele   +9 more
openaire   +2 more sources

Role of CD86 (B7â2) in triggering of antigen-specific IgE antibody response by lipopolysaccharide

open access: yes, 1998
The role of CD86 in triggering of ascaris extract-specific IgE antibody response by lipopolysaccharide was studied. The simultaneous administration of anti-CD86 antibody with ascaris extract and lipopolysaccharide prevented the production of IgE antibody
N Koide   +17 more
core   +1 more source

Expression of Functional Molecule on Plasmacytoid Dendritic Cells Is Associated With HBsAg Loss in HBeAg-Positive Patients During PEG-IFN α-2a Treatment

open access: yesFrontiers in Immunology, 2022
ObjectiveThe ideal endpoint of antiviral therapy in chronic hepatitis B (CHB) patients is to clear hepatitis B surface antigen (HBsAg). This study aimed to evaluate whether the expression of functional molecules on plasmacytoid dendritic cells (pDCs) is ...
Weihua Cao   +16 more
doaj   +1 more source

Rod-like microglia did not take either CD86 or Ym1 staining.

open access: yes, 2013
Iba-1, CD86, MHC-II and Ym1 immunostaining in the NFL. Retinal whole-mounts. In OHT-eyes (A1-A3, C1-C3) CD86 immunostaining (A2, A3) and Ym1 immunostaining (C2-C3: inset) was restricted to few globular cells (arrowhead) located in the NFL and in the ...
Alberto Triviño (499408)   +10 more
core   +1 more source

Late engagement of CD86 after influenza virus clearance promotes recovery in a FoxP3+ regulatory T cell dependent manner. [PDF]

open access: yesPLoS Pathogens, 2014
Influenza A virus (IAV) infection in the respiratory tract triggers robust innate and adaptive immune responses, resulting in both virus clearance and lung inflammation and injury.
Emily K Moser   +2 more
doaj   +1 more source

A subpopulation of KS-positive microglia was CD86-positive.

open access: yes, 2013
The spinal cord sections of SOD1G93A mice at 12, 18 and 24 weeks were stained with KS (A, E, I), M1 marker CD86 (B, F, J), and microglia marker Iba1 (C, G). The merged images are shown in (D, H, K).
Tomohiro Ohgomori (426726)   +15 more
core   +1 more source

Feline CD86 polypeptides and nucleic acids

open access: yes, 2010
The present invention provides isolated and purified DNA encoding feline CD80 (B7-1) ligand, feline CD86 (B7-2) ligand, feline CD28 receptor, or feline CTLA-4 (CD152) receptor, as well as vectors comprising nucleic acid encoding feline CD80, feline CD86,
Winslow, Barbara J.   +3 more
core   +1 more source

Temporal characterization of microglia/macrophage phenotypes in a mouse model of neonatal hypoxic-ischemic brain injury

open access: yesFrontiers in Cellular Neuroscience, 2016
Immune cells display a high degree of phenotypic plasticity, which may facilitate their participation in both the progression and resolution of injury-induced inflammation.
Nina Hellström Erkenstam   +11 more
doaj   +1 more source

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