Results 21 to 30 of about 15,919 (216)

CoQ10 and vitamin A supplementation support voice rehabilitation. A double-blind, randomized, controlled, three-period cross-over pilot study [PDF]

open access: yes, 2019
Objectives: To evaluate the effectiveness of an adjuvant therapy (CoQ10 in its watersoluble form and vitamin A) in supporting voice rehabilitation in a large group of patients with muscle tension dysphonia (MTD).
de Vincentiis, M   +8 more
core   +1 more source

Genetics of Primary CoQ10 Deficiency

open access: yesCurrent Genomics, 2006
Coenzyme Q (CoQ) is a lipophilic component of the mitochondrial respiratory enzyme chain, which transfers electrons to complex III (cytochrome bc complex) from complex I (NADH-CoQ reductase), complex II (succinate dehydrogenase), and from the oxidation of fatty acids and branched-chain amino acids via flavin-linked dehydrogenases.
Naini, Ali   +4 more
openaire   +2 more sources

The potential therapeutic effects of coenzyme Q10 on the sciatic nerve regeneration following short- and long-term injury [PDF]

open access: yesExploration of Neuroprotective Therapy
Aim: This study aims to investigate the effects of administering coenzyme Q10 (CoQ10) after both short-term and long-term sciatic nerve damage. Methods: Six groups of adult male Wistar albino rats were used. Sciatic nerve injury was performed on the rats
Ahmed Omer Mead   +2 more
doaj   +1 more source

Prolonged oral coenzyme Q10-β-cyclodextrin supplementation increases skeletal muscle complex I+III activity in young Thoroughbreds

open access: yesJournal of Applied Animal Nutrition, 2020
Coenzyme Q10 (CoQ10) is an essential component of the mitochondrial electron transport chain (ETC). Decreased skeletal muscle CoQ10 content may result in decreased ETC activity and energy production.
M.F. Rooney   +6 more
doaj   +1 more source

Immunosenescence and CoQ10

open access: yes, 2020
The decline of the immune system associated to aging is becoming a global issue nowadays due to the increase of lifespan. Immunosenescence is the deteriorated adaptative capacity of the immune system leading to increase in morbidity and mortality in elderly population. Aging has been related to a progressive affectation of mitochondria such as increase
Hernández-Camacho, Juan Diego   +2 more
openaire   +2 more sources

The Emerging Role of Disturbed CoQ Metabolism in Nonalcoholic Fatty Liver Disease Development and Progression [PDF]

open access: yes, 2015
Although non-alcoholic fatty liver disease (NAFLD), characterised by the accumulation of triacylglycerol in the liver, is the most common liver disorder, the causes of its development and progression to the more serious non-alcoholic steatohepatitis ...
Elena Bravo   +3 more
core   +3 more sources

CoQ10 Improves Myocardial Damage in Doxorubicin-Induced Heart Failure in C57BL/6 Mice

open access: yesFrontiers in Bioscience-Landmark, 2022
Background: Cardiovascular disease is associated with high morbidity and mortality. Doxorubicin (DOX) is an effective adjunct to cancer chemotherapy but leads to cardiovascular-related side effects.
Zuowei Pei   +6 more
doaj   +1 more source

Anti-oxidants in Parkinson's disease therapy: a critical point of view [PDF]

open access: yes, 2016
Parkinson's disease (PD) is a degenerative neurological syndrome, which is characterized by the preferential death of dopaminergic (DAergic) neurons in the SubstantiaNigra.
Beltramini, Mariano   +3 more
core   +2 more sources

CoQ10 Deficiency: The Lysosomal Paradigm [PDF]

open access: yes, 2022
Coenzyme Q10 (CoQ10) deficiency currently represents the only treatable disorder of the mitochondrial electron transport chain (METC). Generally, in patients with CoQ10 deficiency there is some form of neurological impairment typically presenting as either ataxia and/or seizures.
openaire   +1 more source

Clinical syndromes associated with Coenzyme Q10 deficiency. [PDF]

open access: yes, 2018
Primary Coenzyme Q deficiencies represent a group of rare conditions caused by mutations in one of the genes required in its biosynthetic pathway at the enzymatic or regulatory level.
Alc\ue1zar-Fabra, Maria   +2 more
core   +1 more source

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