Results 61 to 70 of about 1,218 (144)

Emerging concepts of myosin 18A isoform mechanobiology in organismal and immune system physiology, development, and function

open access: yesThe FASEB Journal, Volume 38, Issue 10, 31 May 2024.
This study depicts versatile functions of MYO18A isoforms in transcellular and cytoskeletal signaling processes that modulate epithelial barrier integrity, cellular migration and adhesion, viral infection, endosomal trafficking, macrophage functions, structural development of heart and muscle, and integrity of the alveolar capillary barrier.
William DeKryger, Zissis C. Chroneos
wiley   +1 more source

Proteomic identification of S-nitrosylated Golgi proteins: new insights into endothelial cell regulation by eNOS-derived NO. [PDF]

open access: yesPLoS ONE, 2012
Endothelial nitric oxide synthase (eNOS) is primarily localized on the Golgi apparatus and plasma membrane caveolae in endothelial cells. Previously, we demonstrated that protein S-nitrosylation occurs preferentially where eNOS is localized.
Panjamaporn Sangwung   +5 more
doaj   +1 more source

Intratumor APOL3 delineates a distinctive immunogenic ferroptosis subset with prognosis prediction in colorectal cancer

open access: yesCancer Science, Volume 115, Issue 1, Page 257-269, January 2024.
Our study demonstrated that apolipoprotein L3 (APOL3) is a protective independent prognosticator and yields better responsiveness to both adjuvant chemotherapy (ACT) and targeted therapy in colorectal cancer (CRC). Furthermore, intratumoral APOL3 fostered an immune‐activated tumor microenvironment (TME) and was associated with certain features of ...
Yang Lv   +9 more
wiley   +1 more source

FAP-a and GOLPH3 Are Hallmarks of DCIS Progression to Invasive Breast Cancer

open access: yesFrontiers in Oncology, 2019
Biological markers that could predict the progression of ductal carcinoma in-situ (DCIS) to invasive breast cancer (IDC) are required urgently for personalized therapy for patients diagnosed with DCIS.
Li-Na Yu   +10 more
doaj   +1 more source

Data from Overexpression of GOLPH3 Promotes Proliferation and Tumorigenicity in Breast Cancer via Suppression of the FOXO1 Transcription Factor [PDF]

open access: green, 2023
Zhao-Lei Zeng   +8 more
openalex   +1 more source

Supplementary Figure 3 from Overexpression of GOLPH3 Promotes Proliferation and Tumorigenicity in Breast Cancer via Suppression of the FOXO1 Transcription Factor [PDF]

open access: green, 2023
Zhao-Lei Zeng   +8 more
openalex   +1 more source

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