Results 131 to 140 of about 15,125 (218)

Energy Efficiency and Asymptotic Performance Evaluation of Beamforming Structures in Doubly Massive MIMO mmWave Systems

, 2017
Future cellular systems based on the use of millimeter waves will heavily rely on the use of antenna arrays both at the transmitter and at the receiver.
Buzzi, Stefano, D'Andrea, Carmen
core   +1 more source

LUBAC-mediated M1 Ub regulates necroptosis by segregating the cellular distribution of active MLKL

Cell Death and Disease
Plasma membrane accumulation of phosphorylated mixed lineage kinase domain-like (MLKL) is a hallmark of necroptosis, leading to membrane rupture and inflammatory cell death.
Nadine Weinelt, Kaja Nicole Wächtershäuser, Gulustan Celik, Birte Jeiler, Isabelle Gollin, Laura Zein, Sonja Smith, Geoffroy Andrieux, Tonmoy Das, Jens Roedig, Leonard Feist, Björn Rotter, Melanie Boerries, Francesco Pampaloni, Sjoerd J. L. van Wijk   +14 more
doaj   +1 more source

Nucleic acid sensors and programmed cell death [PDF]

, 2020
Liverpool, Layal, Maelfait, Jonathan, Rehwinkel, Jan   +2 more
core   +2 more sources

Necroptosis of Glycolytic Dendritic Cells Enhances Activation of Gamma Delta T Cells [PDF]

, 2016
Gamma delta (gd) T lymphocytes are a poorly understood class of immune cells that accumulate at sites of inflammation due to infection or autoimmunity. Part of the mystery of this T cell subset derives from lack of knowledge of ligands that bind to the T
Bashant, Kathleen R
core   +1 more source

Saracatinib inhibits necroptosis and ameliorates psoriatic inflammation by targeting MLKL

Cell Death & Disease
AbstractNecroptosis is a kind of programmed cell death that causes the release of damage-associated molecular patterns and inflammatory disease including skin inflammation. Activation of receptor-interacting serine/threonine kinase 1 (RIPK1), RIPK3, and mixed lineage kinase domain-like protein (MLKL) is the hallmark of tumour necrosis factor α (TNF ...
Jingyi Li, Xingfeng Liu, Yuanyuan Liu, Fangmin Huang, Jiankun Liang, Yingying Lin, Fen Hu, Jianting Feng, Zeteng Han, Yushi Chen, Xuan Chen, Qiaofa Lin, Lanqin Wu, Lisheng Li   +13 more
openaire   +3 more sources

MLKL wt Allele [PDF]

, 2020
openaire   +1 more source

ROS via promoting RIP1 auto-phosphorylation to enhance TNF-induced necrosome formation and subsequent necroptosis [PDF]

, 2015
活性氧（ReactiveOxygenSpecies，ROS）的产生存在于生物体的众多生理病理过程中。大量的实验数据表明，活性氧还参与到了细胞凋亡和程序性坏死的进程里。丁基羟基苯甲醚（Butylatedhydroxyanisole，BHA）是公认的活性氧清除剂，异戊巴比妥（Amytal）是线粒体上电子传递到泛醌的抑制剂，二者都曾被广泛报道，可以抑制或者延缓肿瘤坏死因子（TumorNecroticFactorα，TNF-α）诱导的小鼠成纤维细胞系L929的程序性细胞坏死。然而 ...
张荧荧
core  

MLKL et les reliques de la mort

médecine/sciences, 2023
Gwennan André-Grégoire, Julie Gavard
openaire   +1 more source

The neurotoxicant PCB-95 by increasing the neuronal transcriptional repressor REST down-regulates caspase-8 and increases Ripk1, Ripk3 and MLKL expression determining necroptotic neuronal death [PDF]

, 2017
Canzoniero, Lmt, Di Renzo, G, Formisano, L., Guida, N, Laudati, G, Mascolo, L, Molinaro, P, Montuori, P, Serani, A   +8 more
core   +1 more source

Decoding necrosome assembly: harmonizing signal amplification and attenuation through optimal RIP3 stoichiometry. [PDF]

Nat Commun
Li X, Cao Y, Xu F, Zhang Y, Kong Y, Lan C, Zhu R, Lin C, Zhong CQ, Liu Z, Qi H, Huang Y, Xiao Y, Sun GQ, Shuai J, Chen X.   +15 more
europepmc   +1 more source