Results 101 to 110 of about 53,784 (251)

Irisin Ameliorates Hypoxia/Reoxygenation-Induced Injury through Modulation of Histone Deacetylase 4

open access: yesPLoS ONE, 2016
Irisin is a recently identified myokine which brings increases in energy expenditure and contributes to the beneficial effects of exercise through the browning of white adipose tissues. However, its effects in the heart remains unknown. This study sought
Y. Zhao   +5 more
semanticscholar   +1 more source

Flooding: another abiotic stressor to consider in plant-insect interactions

open access: yesFrontiers in Plant Science
Flooding is an increasingly important plant abiotic stress that is intensifying with climate change, yet its ecological implications, particularly for plant- insect interactions remain poorly understood and synthesized.
Satinderpal Kaur, Esther Ngumbi
doaj   +1 more source

Tumor hypoxia and reoxygenation: the yin and yang for radiotherapy

open access: yesRadiation Oncology Journal, 2016
Tumor hypoxia, a common feature occurring in nearly all human solid tumors is a major contributing factor for failures of anticancer therapies. Because ionizing radiation depends heavily on the presence of molecular oxygen to produce cytotoxic effect ...
B. Hong   +5 more
semanticscholar   +1 more source

Reactive species mechanisms of cellular hypoxia-reoxygenation injury

open access: yes, 2002
Exacerbation of hypoxic injury after restoration of oxygenation (reoxygenation) is an important mechanism of cellular injury in transplantation and in myocardial, hepatic, intestinal, cerebral, renal, and other ischemic syndromes.
Li, Chuanyu, Jackson, Robert M
core   +1 more source

Oxygen-Glucose-Deprivation/Reoxygenation-Induced Autophagic Cell Death Depends on JNK-Mediated Phosphorylation of Bcl-2

open access: yesCellular Physiology and Biochemistry, 2016
Background/Aims: The purpose of this study was to investigate the role of autophagy in oxygen-glucose-deprivation/reoxygenation (OGD/R) injury in rat neurons.
Jin Fan   +7 more
semanticscholar   +1 more source

Plant Natural Product Formononetin Protects Rat Cardiomyocyte H9c2 Cells against Oxygen Glucose Deprivation and Reoxygenation via Inhibiting ROS Formation and Promoting GSK-3β Phosphorylation

open access: yesOxidative Medicine and Cellular Longevity, 2016
The opening of mitochondrial permeability transition pore (mPTP) is a major cause of cell death in ischemia reperfusion injury. Based on our pilot experiments, plant natural product formononetin enhanced the survival of rat cardiomyocyte H9c2 cells ...
Yuanyuan Cheng   +3 more
semanticscholar   +1 more source

Mathematical models of repopulation and reoxygenation in radiotherapy

open access: yesThe British Journal of Radiology, 1990
Mathematical models of the effect of ionizing radiation on tumours have not, so far, received general acceptance, and Wheldon (1989) warns of the dangers of over-generalization. Nevertheless, the construction of models may have a certain value, in that we are forced to define our assumptions with clarity. It is interesting to compare two
openaire   +2 more sources

Halothane protects cardiomyocytes against reoxygenation-induced hypercontracture

open access: yes, 1997
BACKGROUND: Resupply of oxygen to the myocardium after extended periods of ischemia or hypoxia can rapidly aggravate the already existing injury by provoking hypercontracture of cardiomyocytes (acute reperfusion injury).
Piper, H. M.   +4 more
core  

Impairment by Hypoxia or Hypoxia/Reoxygenation of Nitric Oxide–Mediated Relaxation in Isolated Monkey Coronary Artery: the Role of Intracellular Superoxide

open access: yesJournal of Pharmacological Sciences, 2011
.: To investigate the effect of hypoxia or hypoxia/reoxygenation on vascular smooth muscle function, mechanical response of monkey coronary artery without endothelium was studied under normoxia, hypoxia, and hypoxia/reoxygenation.
Masashi Tawa   +6 more
doaj   +1 more source

HO-1 Protects against Hypoxia/Reoxygenation-Induced Mitochondrial Dysfunction in H9c2 Cardiomyocytes

open access: yesPLoS ONE, 2016
Background Mitochondrial dysfunction would ultimately lead to myocardial cell apoptosis and death during ischemia-reperfusion injuries. Autophagy could ameliorate mitochondrial dysfunction by autophagosome forming, which is a catabolic process to ...
Dong-Ling Chen   +8 more
semanticscholar   +1 more source

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