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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype [PDF]

Cell Metabolism, 2016
Cellular senescence permanently arrests cell proliferation, often accompanied by a multi-faceted senescence-associated secretory phenotype (SASP). Loss of mitochondrial function can drive age-related declines in the function of many post-mitotic tissues,
Judith Campisi, Arvind Ramanathan, Christopher D Wiley   +2 more
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Chromatin basis of the senescence-associated secretory phenotype

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Inflammation-driven senescence-associated secretory phenotype in cancer-associated fibroblasts enhances peritoneal dissemination

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Senescent Endothelial Cells Sustain Their Senescence-Associated Secretory Phenotype (SASP) through Enhanced Fatty Acid Oxidation

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Fusobacterium nucleatum promotes esophageal squamous cell carcinoma progression and chemoresistance by enhancing the secretion of chemotherapy-induced senescence-associated secretory phenotype via activation of DNA damage response pathway

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Pro- and anti-tumorigenic functions of the senescence-associated secretory phenotype

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