Results 191 to 200 of about 78,621 (245)

Editorial: The interplay between GABA and glutamate in systems physiology and pathophysiology. [PDF]

Front Pharmacol
Rivera C, Lewis SJ.
europepmc   +1 more source

Alzheimer's disease in the <i>Plasticene</i> era: a clinicopathological update on the dual sequestration of amyloid and tau as hijacked innate immune responses. [PDF]

Free Neuropathol
Guth MAS.
europepmc   +1 more source

RETRACTION: Roles of <i>Moringa oleifera</i> Leaf Extract in Improving the Impact of High Dietary Intake of Monosodium Glutamate-Induced Liver Toxicity, Oxidative Stress, Genotoxicity, DNA Damage, and PCNA Alterations in Male Rats. [PDF]

Oxid Med Cell Longev
Cellular Longevity OMA.
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Nitrous oxide and mood: metabolic mechanisms letter in response to Gill et al. [PDF]

EBioMedicine
Reynolds EH.
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Letter to the editor regarding "Inhibition of Mettl3-mediated m6A RNA modification of HMGCS1 protects retinal ganglion cells from glutamate excitotoxicity-induced ferroptosis in a rat model of glaucoma." [PDF]

Int J Surg
Ye L, Hu X.
europepmc   +1 more source

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Role of Mitochondria in the Mechanisms of Glutamate Toxicity

Biochemistry (Moscow), 2005
Current data on glutamate-induced functional and morphological changes in mitochondria correlating with or being a result of their membrane potential changes are reviewed. The important role of Ca2+, Na+, and H+ in the potentiation of such changes is considered.
N K, Isaev, N A, Andreeva, E V, Stel'mashuk, D B, Zorov   +3 more
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Glutamate Toxicity: An Experimental and Theoretical Analysis

European Journal of Neuroscience, 1992
AbstractIn slices of 8‐day‐old rat cerebellum, the lowest concentration of glutamate that induced toxicity (30 min exposure; 90 min recovery) was 100 μM, but the damage only occurred in the outermost regions. As the concentration was raised, the band of necrosis became progressively deeper until, at 3 mM, it was uniform across the slice thickness. At a
Giti, Garthwaite, Geoffrey D., Williams, John, Garthwaite   +2 more
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A Non-Excitatory Paradigm of Glutamate Toxicity

Journal of Neurophysiology, 2002
Retinal ganglion cells are driven by glutamatergic synapses, but they are also very susceptible to glutamate toxicity. Whereas the conventional excitotoxicity model of glutamate-induced cell death requires membrane depolarization, we have found that glutamate toxicity need not be linked with excitation.
Wen, Shen, Malcolm M, Slaughter
openaire   +2 more sources